Elevated triglycerides can be caused by any pre-existing source of excess triglycerides or impaired catabolism. The normal reference range for triglycerides is <1.7 mmol/L (or 150 mg/dl, fasting 12 hours), borderline elevation refers to 1.7 mmol/L-2.3 mmol/L (or 150-200 mg/dl), and elevation refers to ≥2.3 mmol/L (or 200 mg/dl). The etiology of elevated triglycerides can be divided into two main types: primary and secondary. Primary hypertriglyceridemia refers to elevated plasma triglyceride levels caused primarily by genetic abnormalities and usually has a family tendency to run in families. Secondary hypertriglyceridemia refers to an increase in plasma triglyceride levels caused primarily by some disease or external factor and is more common in clinical practice. The causes of secondary hypertriglyceridemia are as follows: 1. obesity: obesity and lack of physical activity, resulting in higher energy intake than energy consumption may lead to increased triglyceride levels. 2, poor dietary habits: excessive carbohydrate intake, as well as heavy alcohol consumption. Increased blood glucose stimulates insulin secretion, which can increase the synthesis of triglycerides by the liver. 3, insulin resistance and diabetes: insulin resistance means that the body cannot effectively use insulin and needs to produce more insulin to maintain normal blood glucose levels. When the condition is further aggravated, diabetes occurs when the blood sugar level is higher than normal. 4, metabolic syndrome: refers to the simultaneous existence of a variety of metabolic abnormalities, including hypertension, obesity, hyperglycemia, etc. 5.Take other drugs: such as diuretics, estrogen, progesterone, steroids, beta-blockers, certain immunosuppressants, anti-HIV drugs, etc. 6, other diseases: including metabolic diseases such as hypothyroidism, Cushing's syndrome, and abnormal liver and kidney function.