Guillain-Barre syndrome is an inflammatory immune disease involving predominantly peripheral nerves. Guillain-Barre syndrome is an autoimmune-mediated peripheral neuropathy, with 60% of patients having gastrointestinal and upper respiratory symptoms, such as diarrhea, 1 to 3 weeks prior to illness. There have been epidemics in rural areas of northern China, where up to 85% of patients were infected with Campylobacter jejuni, and the source of infection was chicken feces. Guillain-Barre syndrome can occur in children and adults, both male and female. Symmetrical progressive weakness of the distal extremities is common. There is also proximal weakness of the extremities that spreads to the distal extremities. Flaccid paralysis and tendon reflexes are diminished or absent. Cerebrospinal fluid examination in patients with Guillain-Barre syndrome suggests protein-cell separation. Electromyography shows delayed or absent F-wave or H-reflex; slowed nerve conduction velocity, prolonged distal latency, and normal or decreased action potential amplitude. When the patient confirms that it is Guillain-Barre syndrome, he should go to the hospital in time for consultation and actively cooperate with the doctor for appropriate treatment.