Keloids are the body’s excessive tissue response to dermal injury, characterized by localized fibroblast proliferation and collagen overproduction. It is closely related to skin injury, patient constitution, and family history. Keloids present as raised dermal lesions that extend beyond the boundaries of the original wound and invade the surrounding healthy skin. Keloids may occur at smaller skin injuries (e.g., ear piercings) or in the absence of a clear inciting stimulus. The pathogenesis of keloids and proliferative scarring is not fully understood, involves a series of wound healing processes, and is influenced by a variety of local and genetic factors. During normal wound healing, there is first a marked local inflammatory response, followed by neovascularization, activation of keratinocytes and fibroblasts at the wound margin, and synthesis of extracellular matrix components. Keloids occur mainly on the upper chest, shoulders, upper back, and head and neck, especially the ears. Keloids can occur as early as 1 month after trauma or inflammation, or as late as 1 year after the precipitating event. Keloids do not shrink on their own, but grow in size over time, and pain and itching are common symptoms. Keloids can be prevented by preventing trauma and infection. If there is any discomfort, it is recommended to seek medical attention.