Troponin includes Troponin I and Troponin T. It is commonly used clinically as a marker to detect the presence of damage or necrosis in the heart muscle. The more common causes of elevation are, first, coronary artery disease, which includes angina pectoris or myocardial infarction. In angina, troponin elevation is mildly elevated, usually in the range of 2-5 fold. If troponin is significantly elevated more than 5 times, it is generally considered to be elevated troponin due to myocardial infarction and myocardial necrosis, which is a serious coronary artery lesion and myocardial infarction due to blockage of blood vessels. Second, heart failure can also cause troponin elevation. In heart failure, troponin elevation can be detected in 75% of patients, which is related to the increased pressure or volume and load of the heart. Third, arrhythmias, rapid tachycardia, such as supraventricular tachycardia, can cause troponin elevation in 37.2% of patients. Fourth, there is a relationship with infectious factors, more commonly viruses or other pathogens that cause inflammation of the heart muscle. Myocardial inflammation can also cause myocardial cell necrosis and troponin release, resulting in a significant increase in troponin when detected. Fifth, pericarditis can also elevate cardiac troponin, especially in young patients and those with recent infectious cellulitis, and the elevation is generally mild. Sixth, endocarditis. Infective endocarditis can also cause elevated troponin, mostly with fever or embolic manifestations, such as stroke, abscesses, or other sites of embolism. Malignant tumors of the heart, as well as aortic coarctation and coronary interventions, can increase troponin to varying degrees.