Usually myocardial bridges do not get worse, but in some cases they may get progressively worse. A myocardial bridge is a congenital condition in which a portion of the myocardium spans the surface of the coronary artery and compresses the coronary artery when the myocardium contracts. If the compression is mild, it does not affect the coronary artery blood supply and the patient may have no obvious clinical symptoms. If the compression of the myocardial bridge is more severe, resulting in relative narrowing of the coronary arteries, it may lead to different degrees of insufficient blood supply to the myocardium, and in severe cases, symptoms of angina pectoris may appear, such as patients may experience corresponding symptoms such as chest tightness, chest pain, dyspnea, and weakness when they are active, exercising, emotionally agitated, or overexerted, and usually this congenital disease does not progress gradually. However, at the site where myocardial bridges occur, coronary atherosclerosis is easily combined, and with age and increased risk factors, atherosclerotic plaques may gradually progress, leading to narrowing of the coronary artery lumen. In this case, due to the aggravation of coronary artery stenosis and the combination of myocardial bridges, it may lead to increased vascular compression in patients, causing myocardial ischemia and inducing angina pectoris. In addition, patients with myocardial bridges may also suffer from other cardiogenic diseases, such as combined hypertrophic cardiomyopathy and hypertension. When myocardial bridge is combined with myocardial hypertrophy, the increased exercise of the patient will cause severe compression of the myocardial bridge or poor perfusion of the myocardial level, thus inducing clinical symptoms such as angina pectoris and chest pain in the patient.