What diseases should I look for to differentiate the diagnosis of myocardial dystrophy from?

Cardiac muscle (cardiacmuscle) is a muscle tissue composed of cardiac myocytes. Cardiac myocytes in the broadest sense include specially differentiated cardiac myocytes that make up the sinus node, intra-atrial bundle, atrioventricular junction, atrioventricular bundle (i.e., the Heath bundle), and Purkinje fibers, as well as general atrial and ventricular myocardial working cells. The first five types of cardiac pacing and conduction system, they contain very few myogenic fibers, or none at all, and therefore have no contractile function; however, they have autonomic and conductive, is the functional basis of cardiac autonomic activity; the latter two have contractile, is the functional basis of cardiac contractile and diastolic activity. Myocardial tissue nutritional disorders occur in cardiomyopathy and so on. Because of the coronary artery multi-branch lesions, even diffuse lesions caused by myocardial ischemia, degeneration, necrosis and fibrosis, myocardial blood supply long-term insufficiency, myocardial tissue nutritional disorders and atrophy, necrosis to fibrous tissue proliferation, which will lead to ischemic cardiomyopathy. 1, dilated cardiomyopathy dilated cardiomyopathy may be and certain factors virus. Bacterial drug toxicity metabolic abnormalities caused by myocardial injury related to, among them, viral myocarditis is considered to be the most important cause. Damage to myocardial tissue from persistent viral infections, autoimmunity including cellular, autoantibody, or cytokine-mediated myocardial injury can cause or induce dilated cardiomyopathy. In addition, perinatal, alcoholism, anticancer drugs, myocardial energy metabolism disorders and neurohormonal receptor abnormalities and other multifactors can also cause this disease. 2.Hypertrophic cardiomyopathy may be related to autosomal dominant inheritance, about 1/3 have obvious family history, cardiac myosin heavy chain and cardiac troponin T gene mutation is the main causative factor. In addition, abnormal catecholamine metabolism, abnormal intracellular calcium regulation, high blood pressure, high intensity exercise, etc. can be used as promotional factors for the development of this disease. Restrictive cardiomyopathy is characterized by endocardial myocardial fibrosis, myocardial stiffness and obstruction of ventricular diastolic filling. It starts slowly, with symptoms such as fever, fatigue, dizziness and shortness of breath in the early stage and total heart failure in the late stage. Atrial fibrillation is also more common, partially combined with visceral embolism. It is mostly found in tropical and temperate regions, and there are only disseminated cases in China.