Colon redundancy is a congenital malformation of the colon, which is caused by overgrowth of the colon due to gene duplication during development. Since colonic redundancy is prone to torsion, obstruction and other complications, the diagnosis can also be confirmed intraoperatively if the corresponding part of the colon is found to be too long during emergency surgery. It is important to note that e-colonoscopy is not used as a diagnostic method for colonic redundancy. Its common complications are intestinal torsion or perforation, intestinal obstruction, intractable constipation, and colonic melanosis. The clinical features of colonic redundancy are functional bowel disease and do not require treatment for those without symptoms. Patients with symptomatic colonic redundancy should be treated conservatively, including diet modification, change of bowel habits, bowel training, and application of gastrointestinal motility promoters such as mosapride, and flora regulators such as rectification. The above treatments can largely relieve the symptoms of patients, especially for those with mild symptoms. For persistent constipation due to colonic redundancy, conservative treatment should also be used first, and about 50% of the patients’ symptoms can also be significantly improved. If the patient’s symptoms are severe and conservative medical treatment is ineffective, surgical treatment is feasible.