NSE is neuron-specific enolase, and lacunar cerebral infarction will lead to elevated NSE.
Cavernous cerebral infarction is a kind of ischemic cerebrovascular disease, which is mainly seen in middle-aged and old people, and the patients may have no obvious clinical symptoms, or they may have mild unfavorable speech, clumsiness of limbs, cognitive function decline and other manifestations, which are related to the site of cavernous cerebral infarction, the size of cavernous cerebral infarction, and the blood supply and other factors.
NSE is present in neural and neuroendocrine tissues, with the highest activity in brain tissue cells, intermediate activity levels in peripheral nerves and neurosecretory tissues, and the lowest values found in non-neural tissues, serum and spinal fluid.
In recent years, the active substances released from nerve cells after cerebral infarction due to ischemia and hypoxia, resulting in damage or disintegration, have attracted the attention of researchers, and among them, neuron-specific enolase has been regarded as a specific and sensitive marker of brain tissue injury.
If NSE is elevated, it is recommended to consult a doctor for a clear diagnosis and standardized treatment under the doctor’s guidance to avoid delaying the condition.