Before answering this question, it is important to understand the trendy term “evidence-based medicine”, which in easy-to-understand language means “medicine speaks with evidence”, and evidence is the facts that we can see. Evidence is what we can see. This fact is not a scientist’s reasoning or hypothesis, nor is it data made in a laboratory with mice, but the result of real clinical use by many people. So which has the evidence, infusions or medication for stroke prevention? The answer is: “There is no evidence for stroke prevention with infusions!” In contrast, there is already a lot of evidence-based medical evidence for long-term prevention of stroke with low-dose aspirin. In a clinical trial published in 2005, nearly 40,000 healthy women over the age of 45 were divided into two groups: one group took 100 mg of aspirin every other day and the other group took a placebo without the drug, and after 10 years it was found that the group taking aspirin had 24% fewer brain infarctions. This study was done on healthy people to prevent strokes, but what is the effect of taking aspirin or other antiplatelet drugs for prevention in patients who already have vascular disease? A summary of the results of 130,000 people who were given the drug found a 25% reduction in brain infarction. With the evidence above we can answer the question that taking medication should be the choice for stroke prevention. But why do so many people still ask for infusion prevention? The idea that infusions prevent strokes is actually misleading, yet it has long been passed down from one person to another. People always think that when infusion is given, the drug enters the blood vessel directly, which can dissolve the blood clot and unblock the blood vessel. In fact, this effect can only be achieved with special thrombolytic drugs within a few hours after the onset of cerebral infarction, and the use of such special thrombolytic drugs requires strict indications, otherwise it will increase the risk of bleeding. And the general infusion has no thrombolytic effect. The lesion we see on the head CT or MRI is already necrotic brain tissue, not a thrombus or embolus blocking a blood vessel, which will not be dissolved by the infusion. After the infusion, the drug will be metabolized by the body soon after it enters the blood circulation and no longer works, while the prevention of morbidity is long-term, just like we brush our teeth every day to maintain the health of our teeth, if we brush our teeth only two weeks or two months a year and not the rest of the time, we will still suffer from dental disease. Therefore, infusion is used to treat emergencies that occur at that time, for example, dizziness, vomiting, numbness and weakness of limbs due to cerebral ischemia, and not as a long-term prevention of cerebral infarction.