Some issues of premature ventricular contractions in children

  1. Is ventricular premature contraction in children dangerous?  Premature ventricular contractions are seen in children with normal heart structure or children with organic heart disease. In organic heart disease, premature ventricular contraction has certain risks, such as myocarditis and cardiomyopathy, which can induce serious heart rhythm disturbances and cause syncope or even sudden death.  Premature ventricular contractions without organic heart disease are called idiopathic premature ventricular contractions and are seen in 10-35% of children with normal heart structure, the incidence of which varies according to the duration of detection and increases with age. The majority of such premature ventricular contractions in children are benign and have a good prognosis.  Much of the so-called experience with premature ventricular contractions is based on the observation of adult cases, however, the adult heart is “resting” or progressively “degenerating”. The child’s heart is in fact a very different heart from the adult heart, and is a developing heart. Many children with ventricular prematurity, especially those of left ventricular origin, may decrease or even disappear as they age.  2. Do children with ventricular prematurity need treatment?  Most ventricular prematureness tends to increase with age in children, and some ventricular prematureness of left ventricular origin may decrease or even disappear as the person ages.In May 2014 the American Academy of Pediatrics and Congenital Electrophysiology (PACES) and the American Heart Rhythm Society (HRS) released the PACES/HRS Expert Consensus Statement on the Evaluation and Management of Ventricular Arrhythmias in Children with Normal Heart Structure, both published online in the Heart Rhythm. The statement concludes that ventricular arrhythmias in children with normal heart structures are mostly benign and often resolve spontaneously without treatment.  Of course, regular follow-up is very important. The main tests are ambulatory electrocardiogram and ultrasound. To understand the regression.  In adults, 24-hour ambulatory electrocardiogram and 2D ultrasound of the heart are used in conjunction with a family history of sudden death, previous history of syncope, organic heart disease, “R on T” electrocardiogram, and the presence of multiform and continuous QRS wave clusters. If the above conditions are present, or if there are more than 10,000 premature ventricular beats in 24 hours, even if there is no family history of sudden death, syncope, organic heart disease, and the presence of R on T, polymorphism, and continuous occurrence of ECG, anti-arrhythmic drugs can be used for treatment. If the above 5 conditions do not occur, the number of premature ventricular contractions in 24 hours is less than 5000, and the patient has no symptoms, no treatment can be used. If there are significant symptoms, antiarrhythmic drugs and anxiolytic treatment may be considered under the guidance of a physician.  In children who have the above mentioned conditions try to treat with nutritional myocardial therapy or with anti-arrhythmic drugs such as metoprolol, cardioplegia, etc. The guidelines for radiofrequency therapy should be more stringent.