Premature ventricular contractions are common clinical arrhythmias, especially in individuals without organic heart disease, and are often detected accidentally during physical examinations or visits for other reasons. Ventricular premature contractions are found in 1% of the normal population using a standard ECG and in 40-75% of the healthy population using a 24-48 hour ambulatory ECG. The incidence of ventricular premature beats increases with age, and in people aged 75-85 years, a single 24-hour ambulatory ECG can record ventricular premature beats in more than 90% of subjects. Of course, in the presence of organic heart disease, such as myocardial infarction, cardiomyopathy, heart failure, mitral valve prolapse, etc. more common, and gender also has an impact on ventricular premature, with organic heart disease in male patients with a 40% higher incidence of ventricular premature than women, the incidence of paired ventricular premature 60% higher. Ventricular premature occurs in everyone’s life, only the age of onset, the number and the accompanying symptoms are different. 2 Misdiagnosis and misdiagnosis are common. In the absence of definite evidence of organic heart disease, ventricular premature contraction in adolescents is arbitrarily attributed to “myocarditis” or “post-myocarditis” without clear diagnostic criteria, and ventricular premature contraction in the elderly is attributed to “coronary artery disease. “This erroneous view is especially true in primary care hospitals. This erroneous view is especially common in primary hospitals, which brings the disadvantage of lack of scientific basis and invariably brings mental and psychological pressure and burden to patients and their families, leading to medical symptoms and a great waste of health resources, and many young people are unable to function normally because of ventricular premature contractions wearing the hat of “myocarditis”. Many young people are unable to go to school or seek medical treatment because of premature ventricular contractions. Because of the misconceptions in the traditional treatment of premature ventricular contractions, it is necessary to re-learn about this common disease. This renewal of concepts has led more clinicians to recognize that idiopathic ventricular premature beats are not necessarily associated with organic heart disease. However, there is another situation in which many physicians “arbitrarily” assume that ventricular premature contractions are benign and lack further management. We have observed clinically that some episodic premature ventricular contractions can lead to syncope, and in such patients, catheter radiofrequency ablation was performed to cure the premature ventricular contractions, with no recurrence of syncope at long-term follow-up. In fact, ventricular premature contractions are associated with a poor prognosis in different types of organic heart disease. Depending on the type and frequency of ventricular premature contractions, the mortality rate is up to three times higher in survivors of myocardial infarction with ventricular premature contractions compared to those without this arrhythmia. Therefore, the risk assessment of ventricular premature contractions is mainly based on: 1. the presence or absence of organic heart disease; 2. the type of ventricular premature contractions; and 3. the combined clinical conditions in a comprehensive assessment: the clinical significance of ventricular premature contractions is not classified solely by their number. There has been little progress in recent years with antiarrhythmic drugs. Current treatment options for ventricular premature contractions focus on improving the underlying lesion by targeting the cause and using antiarrhythmic drugs to reduce the number of preterm contractions, but they cannot cure ventricular premature contractions. Clinical trials have shown that pharmacological treatment of ventricular premature contractions is unsatisfactory regardless of whether they are combined with organic heart disease. It is evident that for the treatment of ventricular premature contractions, other therapies than those targeting the cardiac stroma are needed. The status of catheter radiofrequency ablation as a new treatment for premature ventricular contractions needs to be improved. Catheter radiofrequency ablation has become a routine in the treatment of supraventricular tachycardia, ventricular tachycardia, atrial flutter and other arrhythmias, while the most common clinical arrhythmias —- ventricular premature contractions have not received attention in the treatment. In recent years, with the progress of science and technology, the indications for catheter radiofrequency ablation in the treatment of ventricular premature contraction have been appropriately relaxed, and the status of catheter radiofrequency ablation in the treatment of ventricular premature contraction has been improved by domestic and international guidelines. In clinical treatment, catheter radiofrequency ablation has become a reasonable treatment strategy for frequent or symptom-intolerant or non-benign ventricular premature contractions, but in fact, many of these patients do not receive reasonable and standardized treatment. Therefore, active promotion of standardized treatment of premature ventricular beats has become an urgent task in the treatment of cardiac arrhythmias. Although the catheterized radiofrequency ablation can be carried out in China are large electrophysiology centers, the success rate of catheterized radiofrequency ablation of ventricular premature beats is reported inconsistently in various hospitals, and the efficacy of ventricular premature beats is not certain.