Lumbar disc herniation (hereinafter referred to as LDP), a term used quite frequently by physicians and patients in clinical practice, has almost a tendency to add the symptoms of lumbar pain and imaging reports to LDP without distinction, due to the high incidence of lumbar pain and the unsatisfactory treatment effect, and the widespread use of diagnostic techniques such as CT and MRI in clinical practice, so that the number of patients with LDP has proliferated, resulting in diagnosis and treatment This has led to a proliferation of patients with LDP, resulting in misunderstandings in diagnosis and treatment. Diagnostic criteria for LDP: history of lumbar trauma, chronic strain injury or cold and dampness. Most patients have a history of chronic low back pain before the onset of the disease. It often occurs in young adults. Leg pain is heavier than lumbar pain, and leg pain is typical of the sciatic nerve distribution area. The pain worsens with increased abdominal pressure (e.g., coughing, sneezing). Scoliosis, loss of lumbar physiological anterior convexity, pressure pain in the paravertebral area of the lesion, or radiation to the lower extremities, and restriction of lumbar movement. There is sensory hypersensitivity or dullness in the affected innervated areas of the lower extremities, and muscle atrophy may occur in those with long duration of disease. The straight leg raise or strengthening test is positive, the knee and Achilles tendon reflexes are weak or absent, and the dorsiflexion of the bunion is weak. X-ray radiography: scoliosis, straightening of the physiological curvature of the lumbar spine, narrowing of the vertebral space, and osteophytes at the edges of adjacent vertebrae. CT, MRI examination: it can show the site and degree of disc protrusion. The mechanism of LDP triggering lumbar pain Chronic poor posture or cold causes muscle injury and spasm in the lumbar spine, and the physiological curvature of the lumbar spine becomes backward convex or sideways curved, and the ruptured fibrous ring or nucleus pulposus protrudes backward or laterally with the curvature of the lumbar spine, compressing the nerve roots. Lumbar disc degeneration (thinning and tearing of the fibrous ring, reduction of water in the nucleus pulposus, thinning and calcification of the cartilage end plate, etc.) – narrowing of the vertebral space, misalignment of the posterior joints, stimulation of inflammatory changes in the joint capsule, proliferation of bone redundancy, small intervertebral foramen, and compression of the nerve roots. The lumbar nerve (anterior and posterior branches) is stuck, and the muscles and fascia it innervates are spasmed and blood flow is impaired, causing secondary injury, especially secondary injury to the gluteal muscle, which in turn is stuck to the sciatic nerve trunk, thus aggravating the pain in the lower limbs.