Blood uric acid rises to 600 μmol/L after taking anti-tuberculosis drugs, the main method is to drink more water, promote uric acid excretion, and can be given oral sodium bicarbonate according to the urine PH value. If gout occurs, non-steroidal drugs can be given. If necessary, uric acid-lowering treatment can be given.
Anti-tuberculosis drugs, such as pyrazinamide, have the adverse effect of raising uric acid. During the period of medication, uric acid rises to 600 μmol / L, if the patient does not have cardiovascular and cerebrovascular disease and other related risk factors, can be adjusted in accordance with the medical advice to temporarily do not use uric acid-lowering drugs, you can drink more water to promote uric acid excretion.
At the same time, the application of sodium bicarbonate to regulate the urine PH value to maintain in the range of 6.2~6.9, to reduce the incidence of urinary stones. If uric acid increases and joint pain and other gout manifestations, you can follow the doctor’s prescription oral non-steroidal drugs such as diclofenac sodium and other anti-inflammatory pain.
The increase in uric acid caused by the application of anti-tuberculosis drugs, usually after the discontinuation of drugs, uric acid can gradually drop to normal. If the blood uric acid continues not to decrease or there are risk factors such as cardiovascular and cerebrovascular diseases, you need to receive uric acid-lowering drug therapy.
Eat anti-tuberculosis drugs after blood uric acid do not self-medication or adjust the dose of anti-tuberculosis drugs, need to receive targeted treatment under the guidance of a doctor.