High uric acid alone is usually a symptom of hyperuricemia, which can be treated with drugs that promote uric acid excretion such as benzbromarone, drugs that promote uric acid catabolism such as labrylase, and drugs that inhibit uric acid synthesis such as allopurinol.
Fasting blood uric acid level higher than 420 μmol / L measured twice not on the same day is diagnosed as hyperuricemia. Hyperuricemia only has elevated blood uric acid, without related clinical symptoms, need to control uric acid concentration in a timely manner, so as to avoid the deposition of uric acid gout. Commonly used drugs include drugs that promote uric acid excretion and drugs that inhibit uric acid synthesis.
Drugs that promote uric acid excretion can inhibit the reabsorption of urate in renal tubules, such as benzbromarone. Inhibit uric acid synthesis drugs can inhibit xanthine oxidase, can reduce the production of uric acid, such as allopurinol, febuxostat and so on. Promote uric acid decomposition drugs can catalyze the oxidation of uric acid into water-soluble substances, more easily excreted from the kidneys, such as labrylase, pegloticase, etc.. The above drugs must be used in accordance with medical advice.
Specific drugs need to be adjusted according to the condition, by a professional doctor, avoid self-medication. It is recommended that patients with high uric acid should consult the rheumatology and immunology department in a timely manner, under the guidance of the doctor for standardized treatment and regular follow-up.