Diagnosis: Cerebral palsy is a syndrome caused by non-progressive brain damage from some cause before birth, at birth, or in early infancy. Despite rapid medical development, the prevalence rate has not decreased and is 1-4 per 1,000. The main manifestations are central motor deficits and postural abnormalities, and the symptoms become more pronounced with increasing age. Many patients with cerebral palsy have normal intelligence, but they may also have intellectual backwardness, convulsive seizures, behavioral abnormalities, sensory disturbances and other abnormalities. Although the clinical symptoms may vary with age and brain maturation, the lesions of the central nervous system are non-progressive. Even the same patient with cerebral palsy may have different symptoms in different stages. The diagnosis of cerebral palsy can be determined by visiting the pediatric and neurosurgical departments of the local hospital. Common manifestations: motor development is backward, active movement is reduced; abnormal muscle tone; abnormal posture, in prone position, supine position, pulling from supine to sitting position, standing position, walking, etc., such as scissor leg, pointed foot deformity, horseshoe foot, foot inversion, foot valgus, hip and waist scoliosis, X-like leg; abnormal reflexes. Cerebral palsy can be combined with epilepsy, mental retardation, behavioral abnormalities, sensory perception disorder, speech disorder, microcephaly, joint dislocation and hydrocephalus. Cerebral palsy treatment principles, the main treatment methods available at present Treatment is based on functional training and physiotherapy, combined with drugs, surgery and application of orthopedic devices to reduce symptoms, symptomatic treatment to improve the quality of life of patients. We need to pay attention to early detection and early comprehensive treatment in regular hospitals; comprehensive treatment of movement disorders and comorbidities; combination of home training and doctor’s guidance; functional training according to the law of children’s movement development to promote normal movement development, inhibit abnormal movement and posture, and promote children to produce correct movement in a gradual manner. Early intelligence and language training, motor function training and deformity prevention are crucial. Only normal or near-normal intelligence conditions can make it possible for the child to actively cooperate with effective functional training. Functional training can be done in the rehabilitation department of a regular hospital, where a specific program can be developed through an assessment of the patient’s condition and adjusted in a timely manner according to the recovery situation, mainly consisting of somatic training, skill training, and language training. Medication includes Antan, baclofen, botulinum toxin, and neurotrophic drugs. However, none of them are very effective. Orthopedic devices can promote the physiological alignment of bones, reduce the tension of the muscles around the joints, and also have the effect of inhibiting abnormal reflexes. Physical therapies include hydrotherapy and various electrotherapies such as functional electrical stimulation, transcutaneous electrical stimulation neurotherapy, biofeedback therapy, and high-frequency and ultra-high-frequency currents. About 1/3 of children with spastic cerebral palsy require surgical treatment. Surgical treatment mainly includes neurological surgery, tendon and soft tissue surgery, and bone surgery. The former is for spasticity and the latter is for orthopedics. Neurological procedures include selective posterior rhizotomy (SPR), referred to in some journals as selective dorsal rhizotomy (SDR), and selective peripheral neurotomy (SPN). SPN), with SPR being the most commonly used. In principle, neurological surgery comes first and orthopaedic surgery comes second. After neurological surgery to relieve spasticity, orthopaedic surgery is feasible for patients with residual deformity. Patients with poor results after systematic and regular non-surgical treatment may be considered for SPR surgery if they meet the following conditions: simple spasticity with muscle tone at grade 3 or higher; no obvious fixed contracture deformity or only mild deformity; some motor ability in the spinal extremities; and near normal intelligence, which can cooperate with post-surgical rehabilitation training. Patients are not suitable for surgery if they have the following conditions: low intelligence and cannot cooperate with rehabilitation training; weak muscle strength and hypotonia; tardive dyskinesia, ataxia and torsional spasm; severe fixed spastic deformity of the limbs; severe spinal deformity and spinal instability. SPR surgery is usually performed in the lumbosacral segment of the back or in the cervical region, using electrical stimulation to perform selective posterior spinal nerve rhizotomy, which is called selective posterior spinal nerve rhizotomy (SPR) because it relieves the spasticity more completely while preserving the sense of touch as much as possible, resulting in a significant functional improvement. The efficiency of SPR in the lumbosacral segment for lower limb spasticity is 90%, the functional improvement rate is 75-80%, and the average reduction in muscle tone is about 3 levels. There is also some improvement in upper extremity spasticity and salivation, speech and articulation. The effectiveness of cervical SPR for upper extremity spasticity was 64-83%, with an average reduction in tone of 3 levels. At the current level of testing, because electrical stimulation of the spinal nerve bundle branches does not always correspond to the contraction of the muscle group it innervates, cutting the posterior rootlets can only be a relative release of muscle group spasticity, but it cannot yet target a particular muscle spasm for localized release. In the early postoperative period, abnormal, dull or lost sensation, numbness, pain, deep sensory disturbance, and hypo- or hypertonia may occur. Later in life, abnormal sensation, no relief or recurrence of spasticity, and intractable pain requiring medication control may occur. Limb weakness, bowel and intestinal dysfunction, lumbar spine deformation, spinal instability, and promotion of hip subluxation progression may also occur.