Premature babies have never been the mainstay of pediatric cerebral palsy, which is inseparable from its characteristics. Preterm birth refers to the delivery after 28 weeks of gestation and before 37 weeks of gestation, mostly low birth weight babies, weighing between two and five pounds. The development of the organs of newborns delivered during this stage are immature, and because preterm birth itself is the result of a variety of adverse factors, these adverse factors can also cause damage to the fetus. For example, when the pregnant woman is severely malnourished, suffering from acute and chronic diseases and various high fever diseases, it will cause a shortage of oxygen supply to the fetus. The excessive physical and energy burden of pregnant women, excessive amniotic fluid, multiple pregnancies, as well as uterine fibroids and uterine malformations, can affect the blood flow of the placenta and make the placenta function abnormally, which can cause preterm labor and damage to the fetus. If the birth of a premature baby coincides with a period of rapid brain development and change, it will be more prone to brain injury than a full-term baby. For example, the area around the ventricles of the brain at 26 to 36 weeks is very fragile and vulnerable to adverse factors such as intracranial hemorrhage, which can cause venous blood return obstruction, resulting in venous embolism and secondary brain white matter injury. Premature infants are prone to develop pediatric cerebral palsy with their mothers, for example, preterm infants often do not have adequate nutrition due to feeding difficulties, resulting in a lack of energy for brain development, and the smaller the weight, the higher the chance of cerebral palsy. Premature babies are also prone to diseases such as respiratory distress syndrome and apnea, which can cause respiratory distress and lack of oxygen, and these can be the cause of cerebral palsy.