A lot of pulmonary blood flow is a symptom of tricuspid atresia, and the length of patient survival is closely related to pulmonary blood flow. Those with near-normal pulmonary blood flow can survive for up to 8 years or more; those with a lot of pulmonary blood flow generally survive for only 3 months after birth. Tricuspid atresia is a type of cyanotic congenital heart disease, with a prevalence of about 1 to 5% of congenital heart disease. It is the third most common form of cyanotic congenital heart disease after tetralogy of Fallot and transposition of the great arteries. The main pathologic changes are tricuspid atresia or absence of the tricuspid orifice, patent foramen ovale or atrial septal defect, mitral and left ventricular hypertrophy, and right ventricular hypoplasia. Pulmonary blood flow is a lot of common examination: 1, X-ray examination: tricuspid atresia chest radiographs show a variety of manifestations, orthopantomograms can be seen in the heart shadow is boot-shaped or ovoid, characteristic performance for the right edge of the heart is straight, often not more than the spinal shadow, the left edge of the form of rounded slightly square, the tip of the heart is elevated, the heart of the waist is sunken, if combined with a large blood vessel transposition of the vascular tip is narrower, lateral film shows the heart shadow of the anterior edge of the heart is straight, from the chest wall is farther away from the posterior edge of the heart and the spinal column, the vascular tip of narrowing, large arterial stenosis. The heart shadow can be egg-shaped in patients with narrow vessel tip and misplaced large arteries, and the heart contour can be similar to tetralogy of Fallot in a few cases. In most patients, the heart shadow can be normal or mildly enlarged, with less pulmonary blood flow and reduced pulmonary texture; in a few patients, the heart shadow can be progressively enlarged, with particularly increased pulmonary blood flow and increased pulmonary texture. 2, electrocardiogram: electrocardiogram is characterized by left deviation of the electrical axis, left ventricular hypertrophy, strain, which is very helpful in the diagnosis of tricuspid atresia, 80% of the cases show high or widened P wave with tangential traces, and right atrial hypertrophy is common with markedly hyperexcited P wave, clinically, if you meet cyanotic infants and young children and the electrocardiogram shows left deviation of the electrical axis and left ventricular hypertrophy, you will have a high degree of suspicion of tricuspid atresia because, in tricuspid atresia, 90% have left deviation of the electrical axis, and the left ventricle is hypertrophied. 90% of the cases of tricuspid atresia have left deviation of the electrical axis, and all of them have left ventricular hypertrophy, which is progressively aggravated with the prolongation of time, and the electrical axis of those with thickening of the pulmonary artery is normal or right deviation. 3, echocardiography: echocardiography is the main means of diagnosis of tricuspid atresia, through the two-dimensional echocardiography, can accurately understand the anatomical characteristics of the tricuspid valve, can determine the presence or absence of atrial septal defects and ventricular septal defects and their sizes, but also the size of the atrial and ventricular chambers can be accurately measured. In this disease, the bimodal curve of the tricuspid valve disappears, the four-chamber view fails to see the tricuspid echo-reflex, the interruption of the interatrial septal echo, and the interruption of the upper part of the interventricular septum, and the echocardiography and Doppler examination can see the blood flow from the right atrium to the left atrium and then into the left ventricle, and the amplitude of the mitral valve activity increases, and the right atrium, left atrium, left ventricular cavity are all enlarged, and the right ventricle is small or disappears. In addition, this test can be used to understand the relationship between the left and right ventricular chambers and the great vessels, to determine the presence or absence of stenosis of the pulmonary and aortic valves, as well as to diagnose the presence or absence of comorbid malformations, such as arterial ductus arteriosus, narrowing of the aorta, aortic hypoplasia, dissected aortic arch, aortic atresia, and so on.