One month after the blocker is implanted in the heart, the body’s own endothelial cells cover its surface, and after 3 months the endothelial cells completely encapsulate and fix the blocker. Therefore, we require children to avoid strenuous exercise for 3 months after the intervention for precordial disease. After more than 10 years of follow-up observation, patients with congenital heart disease treated with interventional therapy are completely restored to health, their growth and development are not affected, and they are able to work, study and live like healthy people; marriage and childbirth are not affected. Common complications of interventional treatment are as follows: intraoperative: when blocking the larger internal diameter of the unclosed arterial catheter, if the blocking umbrella is repeatedly released and retrieved several times, it may cause pulmonary artery entrapment; for severe pulmonary valve stenosis, spasm of the right ventricular outflow tract may easily occur during balloon expansion, which may cause aggravation of hypoxemia in severe cases; when performing balloon pulmonary valvuloplasty and interventional treatment of ventricular septal defect, it may In some children, the blocker is found to be dislodged immediately after surgery, before they get off the operating table. The current state of the art has eliminated the possibility of serious complications such as cardiac rupture. Postoperatively, complications such as dislodgement of the blocking device, residual shunt and hemolysis, which occurred in the early stage of arteriovenous catheter occlusion, have been decreasing; in the past few years, we found that a very small number of cases after the placement of atrial blocker could cause perforation of the top of the left or right atrium, resulting in pericardial pressure and subsequent hemodynamic effects, and a very small number of patients had aortic perforation, and sometimes acute hemolysis could appear in the weeks after surgery. The perforation of the left atrium and aortic root is now thought to be related to the erosion of the atrium and aortic wall by the atrial defect occluder. It should be noted that atrial defect closure is safe and the risk of erosion of the plugger is minimal, with an incidence of 0.1%, occurring in patients with anterior or anterosuperior shortage of the atrial defect, with erosion occurring at the top of the left or right atrium, and the need for regular follow-up after intervention should be emphasized.