Fewer patients with SLE may develop serious intraocular complications. Immune complex deposits are seen in the choroidal capillaries, the basement membrane of the ciliary body, and the bulbar conjunctiva. The retinopathy of this disease may result from two separate mechanisms: vascular embolism caused by antiphospholipid antibodies, which can be effectively treated with anticoagulants, and immune-mediated vasculitis, which is most effectively treated with immunosuppressive methods. This disease can affect both small arteries and small veins. This embolic disease in SLE is characterized by cotton wool-like spots with or without intraocular hemorrhage. Jabs and colleagues reported on 11 patients with retinal vasculitis. Most patients (55%) had a poor visual outcome (20/200 visual acuity or less) and a high incidence of CNS disease (73%). Vascular blocking lesions such as central retinal vein block may be present, and neovascularization is often seen in cases of extensive blockage of small arteries as well as secondary ischemic lesions.