Chronic blood pressure of 160/100 mmHg may lead to cerebral hemorrhage, and transient blood pressure of 160/100 mmHg usually does not lead to cerebral hemorrhage.
The most common cause of cerebral hemorrhage is hypertension combined with fine arteriosclerosis. The main pathogenesis of hypertensive cerebral hemorrhage is the rupture of small arteries in the brain due to chronic lesions under the effect of long-term hypertension.
The intracranial arteries are characterized by the lack of middle layer of muscle cells and outer layer of connective tissue and the absence of outer elastic layer. Long-term high blood pressure can make the small cerebral arteries undergo vitreous degeneration, fibrinoid necrosis, and even the formation of micro-aneurysms or entrapment aneurysms, which can easily lead to vascular rupture and hemorrhage when the blood pressure suddenly rises on the basis of this.
Therefore, long-term blood pressure 160/100mmHg, affecting the small cerebral arteries, can lead to cerebral hemorrhage, if there is no history of hypertension, and a transient blood pressure 160/100mmHg, generally does not lead to cerebral hemorrhage.
Long-term blood pressure 160/100mmHg, there is a risk of cerebral hemorrhage, and this blood pressure itself will occur in the heart, brain, kidney and other target organ damage, it is recommended that the regular in the cardiology department to control blood pressure.