Etiology and pathogenesis: Foramen ovale detachment is a pathological process in which the vitreous and retina are jointly involved. As a result of retinal atrophy and degeneration or vitreous traction, the retinal neuroepithelial whole-layer fissure is formed, and the vitreous traction on the edge of the fissure, and the vitreous liquefied by degeneration enters the subretina through the fissure to form retinal detachment. Clinical manifestations: High myopia, aphakic eyes, retinal lattice-like degeneration and history of ocular trauma are prone to retinal detachment. Patients have symptoms such as floating objects, flashing sensation or curtain-like blockage in front of the eyes at the beginning of the disease. As the detachment expands to the macula, vision decreases to varying degrees until only light perception remains. The detached retina is grayish-white and undulating, and all of them have retinal fissures. Diagnosis: The diagnosis of foraminal retinal detachment is not difficult based on the patient’s complaints and the fundus of the eye. Ultrasound scan also helps in the diagnosis. Treatment: Early surgical treatment is recommended for pore-derived retinal detachment, including scleral buckling and vitreous surgery. The visual prognosis is closely related to whether the macula is detached before surgery and the length of detachment. If the macula is not detached or detached for less than 1 week, it is expected to recover better vision after surgery, but if the macula is detached for more than 1 month, it is not easy to fully recover vision after surgery. (This article is authorized by Dr. Tian Bei.)