The retina is a translucent membrane in the eye, a nerve tissue that senses external light information, and is located at the base of the eye, functioning as the equivalent of a camera negative. Pore-derived retinal detachment is a more serious and common blinding eye disease. It is caused by a fissure in the retina, where the liquefied vitreous enters the subretina through the fissure, causing the retina to bulge out of its normal position. After retinal detachment occurs, the nutrition of photoreceptor cells is damaged, and if it is not reset in time, the retina will undergo atrophy and degeneration, resulting in irreversible damage to visual function. Moreover, long-term retinal detachment will lead to proliferative vitreoretinopathy (PVR), which makes treatment more difficult and less successful, and may also lead to secondary uveitis, pupillary atresia, concurrent cataract, and even eye atrophy. Therefore, early diagnosis and early surgery are necessary to save vision and the eye as much as possible. The current effective treatment for foraminogenic retinal detachment is surgery. The mainstream surgical procedures are scleral buckling (also known as retinal condensation, extra-scleral silicone pad compression) and vitrectomy. Both surgical procedures have their own advantages and disadvantages, and have their own indications (i.e., their respective applicable conditions or ranges), which need to be chosen by the doctor according to the patient’s specific situation. What the surgeon can do is to provide the opportunity for the retina to return to its normal position through appropriate surgery, but the patient’s cooperation is also needed to maintain a certain head position after surgery. About 95 percent of retinal detachments can be cured by surgery, but a few of these patients may require two or more surgeries. Other patients may not be cured despite multiple surgeries, or may be too ill or delayed for the surgery to be worthwhile. Cure means that the retina is repositioned, that is, the retina is anatomically restored to its normal position and is no longer detached. Recovery of vision after surgery, however, is a relatively difficult problem, and there are no medications available to promote recovery of vision. The degree of vision recovery is related to the site and duration of the retinal detachment. Generally speaking, if there are no other factors causing vision loss, within 1 week of macular detachment, about 75% of patients can recover more than 0.4; between 2-8 weeks, about 50% of patients can recover more than 0.3; more than 8 weeks, vision recovery will be very difficult, and 0.1 is a relatively good result. If the area of retinal detachment does not involve the macula, about 85% of patients can maintain their preoperative vision after surgery. About 15% of patients will have lower vision than the original vision due to macular edema, macular anterior membrane, concurrent cataract, glaucoma, etc., and may need medication or surgery again. After a retinal detachment has been surgically reset, about 10% of patients will have another retinal detachment in the same eye during their lifetime, and the risk of having a retinal detachment in the other eye is also about 10%. Therefore, after retinal repositioning, it is usually necessary to visit the hospital every 6-12 months for detailed examination of the retina with dilated pupils. If lesions such as retinal fissures or lattice-like degeneration areas can be found, these dangerous lesions can be closed with laser retinal photocoagulation to reduce the risk of retinal detachment. If there are floating objects in front of the eyes or an increase in the existing floating objects or a flashing sensation in front of the eyes, you need to go to the hospital as soon as possible for dilated pupil examination of the fundus, which are the precursors to the occurrence of retinal fissures or detachment. If you are unlucky and a retinal detachment occurs between reviews, you will have to undergo surgery.