Addy’s disease is primary hyperaldosteronism. Due to adrenal tuberculosis or autoimmune reaction, the adrenal cortex is damaged, resulting in low adrenal cortical function, called Addy’s disease. The main clinical manifestations of Addy’s disease are as follows: First, the manifestation of glucocorticoid deficiency, the adrenal cortex bundle is mainly secrete glucocorticoid, when appearing Addy’s disease, the symptoms of glucocorticoid deficiency can appear, such as fatigue and weakness, low blood sugar, poor resistance, easy to catch cold, infection, poor stress ability of the body, etc. Second, the symptoms of salt corticosteroid deficiency, the adrenal cortex bulbous zone to secrete aldosterone, aldosterone deficiency will appear when Eddie is sick, can be manifested as low blood pressure, salty food, postural hypotension and other symptoms. Third, primary hyperaldosteronism, can appear skin, mucous membrane, areola and other friction parts of hyperpigmentation. Fourth, if hypoadrenocorticism is not treated in time, it can occur under the causative factors such as infection, vomiting and diarrhea, and adrenocortical crisis can occur, endangering patients’ lives.