Alcoholic tremor is a common clinical symptom of alcoholic cerebral atrophy. Alcoholic cerebral atrophy (ACA) refers to the irreversible reduction of brain tissue due to chronic alcohol consumption. It is controversial whether this disease constitutes a separate clinicopathologic disorder. Some authors consider alcoholic cerebral atrophy to be a pathologic concept, a term first derived from cerebrographic studies. It specifically refers to relatively young alcoholics, with or without clinical signs of cerebral pathology, who have enlarged lateral ventricles and widened frontal sulci on encephalography. Modern imaging has corresponding findings. The etiology of alcoholic tremor has confirmed that large amounts of uncontrolled alcohol consumption have a significant detrimental effect on many organ systems of the body, and the nervous system is one of the main target organs for the pathogenesis of alcohol abuse. The nervous system is one of the main target organs of alcohol abuse. Another common cause of alcoholic tremor is the alcohol withdrawal syndrome (ATS). Acute alcohol withdrawal syndrome occurs when a person who has been drinking heavily for a long period of time (more than 2-3 weeks) suddenly stops drinking or reduces the amount of alcohol he or she consumes, and the patient then develops a series of symptoms and signs, which is known as alcohol withdrawal syndrome or detoxification syndrome. The pathogenesis is due to the loss of the inhibitory effect of alcohol in the central nervous system, resulting in over-excitation of the cerebral cortex or β-adrenergic nerves. It occurs in alcoholics who are already physically dependent. The syndrome is characterized by tremors, delirium, convulsions, confusion, and psychomotor and autonomic hyperexcitability. Each of the main symptoms of the withdrawal syndrome can occur in a more or less pure form, but often in different combinations.