The views in this article are only personal opinions. Many patients, often ask me a question whether polycystic ovary syndrome can be cured, because they see many experts, doctors, articles or some literature, most of them think that polycystic ovary syndrome cannot be cured. In this regard, I have a different view. Polycystic ovary syndrome can be completely reversed or cured. First of all, in the population of women, polycystic ovary syndrome is less common in adult women, especially in those who are 30 or 35 years old or older. Polycystic ovary syndrome can also be seen less frequently in some cases of polycystic ovary syndrome after childbirth, when the reproductive endocrine regulatory axis functions and the reproductive organs and target glands mature. The occurrence of polycystic ovary syndrome should be mainly related to nutritional overload and the current dietary structure. In particular, the combination of additives and hormones in various types of food, as well as the psycho-psychological aspects due to excessive intake, has resulted in a substantial increase in the occurrence of polycystic ovary syndrome compared to 10 or 20 years ago. The occurrence of this disease, like hypertension, diabetes mellitus, atherosclerosis, etc., is related to life and intake and is the result of a multi-causal, multi-linked pathological change. Therefore, at its core, polycystic ovary syndrome remains a reproductive endocrine disorder with gonadal (ovarian) dysfunction as the main cause. The important features of the pathology of polycystic ovary syndrome are decreased LH (luteinizing hormone) sensitivity in the ovary, defective LHR (luteinizing hormone receptor) or reduced synthesis, and disruption of the aromatase pathway, which results in excessive synthesis of androgens that cannot be further aromatized into estrogens. In combination with metabolic syndrome, polycystic ovary syndrome may be characterized by insulin resistance or hyperinsulinemia, excessive social nutrition, excessive energy intake, especially raw materials for sex hormone synthesis, excessive cholesterol intake, increased pancreatic and metabolic burden, enhanced anabolic effects, and obesity, which aggravate local ovarian dysfunction. The treatment of polycystic ovary syndrome, improving ovarian endocrine function is the key and improving metabolism is the foundation. In the treatment of polycystic ovary syndrome, improving the endocrine function of the ovary is the key, and improving the metabolic status is the foundation. In polycystic ovary syndrome, the ovarian peritoneal fibrosis and the non-rupture of the follicle mostly occur during ovulation promotion therapy, which is related to high LH and low female or Kaohsiung state, and mainly related to the thickening of the follicular peritoneal fibrosis. If the pregnancy is successful and the pregnancy is completed in October, the ovaries have also acquired the best and resting process. The reproductive function matures and becomes perfect, and the chance of reoccurrence of polycystic ovary syndrome is greatly reduced, although if one is still overly obese, obese or insulin-resistant polycystic ovary syndrome can still occur. Overnutrition, weight gain, obesity, hyperinsulinemia, insulin rejection (abnormal receptors), and abnormal glucose tolerance are the underlying causes of polycystic ovary syndrome. These processes, however, can be completely and effectively stopped or reversed by healthy and rational control of multiple aspects of diet structure, exercise, and lifestyle, the earlier the better the advice. Therefore, attention and clear understanding of the disease and its pathophysiological basis can reduce blindness and is important for countermeasure development. In many infertile, unmarried or adolescent women, the occurrence of polycystic ovary syndrome is mainly related to long-term anovulation, immature ovarian function and unstable and immature gonadal axis cycle regulation mechanisms. During pregnancy and postpartum time, excessive weight gain leading to obesity and excessive energy intake can increase the risk of recurrence of polycystic ovary syndrome after delivery, as well as increase the risk of insulin refusal and diabetes. Proper diet, control of excessive intake and prevention of metabolic syndrome are important foundations for the prevention of polycystic ovary syndrome, diabetes and cardiovascular disease. In prolonged anovulation, many women can have polycystic ovarian changes, but they are not diagnosed with polycystic ovary syndrome. Prolonged menstrual disorders, scanty periods and anovulation can progress to the more complex polycystic ovary syndrome. This leads to increased difficulty in restoring the menstrual cycle and ovulation. Stable ovulation, regular endocrine regulation, or a successful pregnancy are the better outcomes for polycystic ovary syndrome. Successful pregnancy is also the best treatment for polycystic ovary syndrome. No drug can compare with an October pregnancy in that one aspect further intensifies the maturation of ovarian function, another aspect makes the ovary fully resting, three aspects increase the development of the ovary and the synthesis and refinement of various receptors, four aspects improve the blood supply to the ovary and completely stop the follicular peritoneal fibrosis and instead facilitate the degradation of the stroma or collagen, and five aspects completely reverse the local inflammatory fibrotic process of the ovary. However, by no means all problems are solved when a woman with polycystic ovary syndrome becomes pregnant. If she remains obese, insulin refusal and metabolic syndrome during pregnancy and postpartum, all are prone to recurrence and treatment difficulties. Adequate transformation of the endometrium is also very important. In many cases of polycystic ovary syndrome, Kaohsiung, low estrogen or both are present. Promoting the production of ovarian-derived estrogen is one of the important steps in treatment. In my clinic, I usually use herbal methods to promote follicle development. In some cases, menstruation comes sooner, sometimes it takes longer, mainly to promote follicle development from the beginning; in some cases, after a long period of amenorrhea, ovulation occurs again through herbs, and only after that, menstruation comes. Caution is also needed when treating polycystic ovary syndrome with ovulation promotion. One aspect is to avoid LUFS (follicular unruptured luteinization syndrome); a more important aspect is to evaluate the ovaries before ovulation promotion and to use the process of ovulation promotion in parallel to avoid the occurrence of OHSS (follicular hyperstimulation syndrome). Polycystic ovary syndrome is a relatively complex disease and treatment requires a combination of multiple aspects, and simple methods are often difficult to achieve expectations. There should also be an evaluation regarding the use of Daimler 35 and MaFuLong. And they should not be used in a general way. Especially in the case of unmarried or adolescent women or postpartum menstrual disorders and polycystic ovary syndrome, the use of Daing 35 or MaFuLong should be evaluated comprehensively and used with caution. Excessive suppression of the gonadal axis does not yield the desired rebound effect. Chinese medicine treatment, which is effective, reasonable grouping, especially in-depth evidence rubbing, in the regularity to be followed, has an important role in the improvement of the polycystic ovary syndrome syndrome comprehensive state, the success of follicle development, the success rate of ovulation, and the increase of conception rate. The combination of Chinese and Western medicine is unique in the treatment of polycystic ovary syndrome. It should be taken seriously and relevant norms should be developed. There are various diseases that can manifest as symptoms of polycystic ovary syndrome, especially when Kaohsiung is present, which requires differential diagnosis and requires physicians to have knowledge in many aspects, especially in the discipline of Western medicine endocrinology. Without a clear and explicit diagnosis and accurate grasp of the pathophysiological state, blind treatment, in some cases, does not yield better expectations. In endocrine therapy, the knowledge related to modern pharmacology and even molecular pharmacology, the knowledge of pharmacology of herbal components, and the knowledge and progress related to molecular pathophysiology are important guidance for the treatment of polycystic ovary syndrome. Having this knowledge can further reduce the blindness and ambiguity in the treatment process of doctors. Blind belief in some prescriptions or lack of in-depth pathophysiological analysis and judgment, and just purely herbal prescription treatment, sometimes have slower efficacy. Follicular development and dominance, as well as the relationship between multiple links with endocrine, metabolic base state, and overall state improvement need to be clear, orderly, and gradually reversed, which is difficult to achieve results in a short time. During the treatment process, it is equally important for the doctor to be very clear and for the patient to have a general understanding of the program flow, for cooperation and long-term treatment.