Aseptic redundancies are most commonly found in areas of turbulence, scarring (e.g. after infective endocarditis) and areas of endocardial damage due to intra- and extra-cardiac factors. So, what should be done to prevent aseptic flab? The following is a detailed description: Prevention of aseptic flaccidity should be actively prevented and treated for primary diseases. Patients who are old and frail, have chronic wasting diseases, malignant tumors and other diseases should be carefully observed clinically for early detection and early treatment. Non-infective valvular vegetation (NIVV) in addition to NBTE, the following conditions are common: 1, systemic lupus erythematosus (SLE): as early as 1924 Libman and Sacks first reported that SLE can generate superfluous in the heart valve recent echocardiography confirmed that SLE In recent years, echocardiography has confirmed that 18% to 40% of patients with SLE have a combination of NIVV called Libman-Sacks endocarditis. The mitral valve is most susceptible to involvement, followed by the aortic valve superfluous pathological features are generally 3-5 mm in size, gray or pink, pea-shaped or flattened, often adhered to the endocardium, may be accompanied by thrombus, microscopic fibrin, fibrous tissue formation may have platelet or thrombus adhesion, with a small number of mononuclear cell infiltration. (1) clinical manifestations: the main manifestation is the symptoms of SLE, a few cases can appear mitral valve closure incomplete systolic murmur, if involved in the aortic valve can produce aortic regurgitant murmur, occasionally flabby dislodgement can also cause the corresponding signs of body artery embolism a group of 50 cases of SLE autopsy found that there were 10 cases of cerebral infarction, 5 of which were associated with Libman-Sacks endocarditis flabby The obstruction was associated with a Libman-Sacks endocarditis bulge. Echocardiography, especially esophageal echocardiography, is more likely to detect valvular redundancies and has diagnostic value, but sometimes it is difficult to differentiate from SLE combined with SIE. If the plasma phospholipid level is increased and there is antigenic antibody reaction, it is most likely that SLE is complicated by Libman-Sacks endocarditis. (2) The main treatment of the primary disease, the larger superfluous some advocate the use of anticoagulation therapy commonly used are warfarin, vinblastine coumarin, but need to consider the risk of bleeding, its efficacy has yet to be further confirmed. If the involvement of the valve leads to severe closure insufficiency, surgical treatment is required, but this case is rare. 2, primary antiphospholipid syndrome (primary antiphospholipide syndrome, PAPS): this syndrome is associated with the presence of phospholipid antibodies in the serum, patients often have recurrent miscarriages, thrombocytopenia and recurrent arteriovenous embolism as a feature. If the prolonged prothrombin time is not corrected by adding normal plasma, PAPS should be considered as a possible diagnosis. Valvular redundancy in this disease is mainly detected by echocardiography, and the size of the redundancy is usually 2 to 6 mm.