Hepatic stasis is mainly caused clinically by diseases such as Buga’s syndrome, constrictive pericarditis, and severe right heart failure. In this case, the central vein of the hepatic lobule and its adjacent hepatic sinusoids are highly dilated and stagnant, and the hepatocytes atrophy or even disappear due to hypoxia and stress. Severe hepatic stasis may also cause hepatocyte necrosis, while the hepatic sinusoids in the peripheral part of the hepatic lobules are less stagnant and hypoxic, and hepatocytes may show varying degrees of steatosis. Multiple fat vacuoles may also be seen in the hepatocytes of the peripheral portion of the hepatic lobules. The lesions can cause an increase in the size of the liver and therefore can irritate the hepatic pericardium and present with signs and symptoms of pain in the liver area as well as tenderness. The hepatocellular damage may also cause hepatic decompensation and impairment, and long-term chronic liver stasis may eventually lead to further aggravation of liver fibrosis and hardening of the liver, eventually leading to the formation of cirrhosis. The clinical treatment of liver stasis may vary depending on the cause of liver stasis.