Unilateral gluteus medius paresis is characterized by a backward tilt of the trunk, which is called a chest and belly gait; unilateral gluteus medius paresis is characterized by a lateral bending of the trunk to the affected side in the supporting phase, with the shoulder of the affected side restraining the other side of the pelvis from descending; bilateral gluteus medius paresis is characterized by an alternating lateral bending of the trunk to the supporting side, which is called a duck gait; hip adductor spasm is characterized by a crossed gait; hip internal rotation paresis is characterized by a more severe external rotation of the lower limb on the affected side in the supine position than on the other side, and an inability to return to the neutral position on its own The cause of hip flexion deformity is poor abdominal muscle function, but it may also be compensated by quadriceps palsy, posterior calf muscle group palsy, or hip flexor spasm; the cause of circle deformity in the stride phase is poor centripetal contraction of the lower extremity flexors or extensor spasm; quadriceps palsy is manifested by poor weight-bearing function and knee hyperextension on the affected side; poor knee flexion in the stride phase is caused by spasm of the knee extensor muscle group or centripetal contraction of the knee flexor muscle group Poor function of the knee extensor group spasm is associated with poor function of the posterior calf group muscles. This deformity is also characterized by a lack of knee flexion compensated by hip flexion; inversion of the knee when the foot is biased medially in the stride phase, which means that the external knee muscles cannot balance the internal rotation muscles; and valgus of the knee when the foot is biased laterally, which means that the internal rotation muscles are relatively poor. The spasm of the knee flexor muscle group can lead to abnormal knee flexion, which affects standing; tibialis anterior muscle paresis is manifested by foot droop and difficulty in foot contouring, if accompanied by hip flexion compensation, it is manifested by straddling the threshold step, and in mild cases, it is also manifested by gravity cushioning phase with sound of palm tapping; triceps calf paresis is manifested by weakness of backward stirring of the affected foot and difficulty in body forward propulsion, which is manifested by compensation of trunk forward leaning, shortening of stride length on the other side, delay in heel leaving the ground, difficulty in standing on the forefoot when the affected leg is loaded on one leg, and difficulty in jumping on one leg. Difficulty in jumping on one leg, difficulty in going downhill (stairs), and inability to run on the toes; triceps spasm in the lower leg manifested as horseshoe foot (often accompanied by forefoot inversion), difficulty in foot contouring, ankle clonus, mostly accompanied by hip flexion compensated for as a straddle step; inversion deformity of the foot is caused by relatively strong function or spasm of the inversion muscle group, also related to insufficient internal rotation of the knee, spasm of the posterior calf muscle group, and paralysis of the toe extensors; inability of the foot and ankle to provide stable support for movement manifested Difficulty in forefoot weight-bearing is manifested by inability to stand or maintain a single-leg forefoot station, difficulty in running, difficulty in jumping on one leg, difficulty in walking up hills, etc., and is associated with paralysis of the triceps, bunions, and internal foot appendages; difficulty in bunion flexion may be manifested by hyperextension of the thumb (e.g., Babinski’s sign), weakness of the backstroke of the affected foot, etc.; difficulty in toe dorsiflexion may be manifested by the remote portion of the foot wrapping around the Difficulty in standing up, especially from a low stool, is due to the inability to shift the center of gravity forward, poor centripetal contraction of the extensor muscles of the lower limb, and failure to raise the back in time. Difficulty in walking upstairs is caused by inadequate knee flexion, foot drop, poor weight-bearing ability on the affected side, and inadequate forward shift of the center of gravity due to hip flexion; difficulty in walking downstairs is caused by knee flexion due to knee hyperextension due to hip flexion, inadequate forward shift of the center of gravity, poor control of tibial advancement, and inadequate relative length of the lower extremity in the stride phase due to excessive hip flexion; difficulty in running is caused by the posterior calf group of muscles in supporting the relative ankle. poor control of the posterior group of muscles in supporting the relative ankle making it difficult to maintain ankle plantarflexion, poor synergistic swing function of the upper limb; difficulties in single leg jumping in the affected leg due to paralysis or poor explosive power of the posterior group of muscles of the lower leg and the internal foot appendages, weakness of the thumb flexors and spasm of the toe flexors.