There is a relationship between high blood pressure and urine protein, and often patients with long-term hypertension will show an increase in urine protein after they have kidney function damage. Under normal circumstances, the urine does not contain protein, when the kidney function is reduced, manifested as increased urine protein, long-term hypertension, especially in patients with poor blood pressure control, prone to hyperalgesia. Long-term hypertension will lead to the gradual proliferation and thickening of glomerular arteries, followed by atherosclerotic changes, which will lead to reduced renal blood flow over time, and then hyperalgesia will occur. After the kidney function decreases, the filtration of the kidney will be reduced, so the urine protein increases. Once hypertensive patients have increased urine protein, it is important to control blood pressure in a timely manner, to strictly control blood pressure at 130/80mmHg, which can delay the deterioration of kidney function.