Fibrous exudate contains a large amount of cellulose as the characteristic exudate. The large amount of fibrin exudate suggests that the capillaries and small veins are more damaged, the permeability is significantly increased, and a large amount of fibrinogen exudes out of the blood vessels, which is converted to fibrin under the action of tissue factor released from the necrotic tissues. How to check for fibrous exudate? When fibrinitis occurs in the plasma membrane and lungs, a small amount of fibrin exudate, can be dissolved and absorbed; a large amount of fibrin exudate is prone to mechanization, and even plasma membrane cavity occlusion, causing organ dysfunction. Such as fibrillar pericarditis, due to the heart’s beating, the pericardium of the dirty wall of the two layers of friction, so that the fibrin exuded in the pericardial cavity of the pericardial membrane surface in the form of fluffy, known as “fluffy heart”. If the neutrophil exudes less, the protein hydrolase released is relatively insufficient to dissolve and absorb the cellulose completely, it can be mechanized through the growth of granulation tissue and finally lead to fibrosis. If the fibrosis occurs in the pleura, it may cause thickening and adhesion of the pleura, or even occlusion of the pleural cavity. When it occurs in the lungs, such as the gray hepatoid stage of lobar pneumonia, there is a large amount of fibrin exuding from the alveolar cavities, making the lungs solid. Fibrinous inflammation is dominated by the exudation of fibrinogen, followed by the formation of fibrin, or fibrin. Fibrillitis tends to occur in mucous membranes, plasma membranes, and lung tissues and needs to be differentiated. 1, occurs in the mucosa, exuding fibrin, necrotic tissue and neutrophils together to form a pseudomembrane, also known as pseudomembranous inflammation. Diphtheria pseudomembranous inflammation, if it occurs in the pharynx is not easy to fall off called solid membrane inflammation. 2. If it occurs in the trachea, it can be easily detached as floating membrane inflammation, which can easily cause suffocation. 3.Fibrinous inflammation of the plasma membrane can cause fibrinous adhesions in the body cavity, followed by fibrinous mechanization and fibrous adhesions. 4, occurs in the lung, in addition to a large number of exudate fibrin, can also be seen in a large number of neutrophils, common in lobar pneumonia.