The etiology of aortic coarctation is still not well understood. Hypertension is the most important factor predisposing to aortic coarctation, with 70% to 90% of patients with aortic coarctation having elevated blood pressure, and about half of proximal and almost all distal aortic coarctation having hypertension; degenerative degeneration of the middle layer of the aorta such as Marfan syndrome, Ehler-Danlos syndrome, congenital Aortic stenosis, bilobed aortic valve and other genetic defects in connective tissue can easily lead to intimal rupture and hematoma formation; other medical trauma such as atherosclerosis, aortic trauma, inflammation, arterial intervention, valve replacement and other medical trauma can also be the cause of aortic coarctation. Pathogenesis of aortic coarctation The pathogenesis of aortic coarctation is due to a decrease in the adhesion between the intima and the middle layer of the aorta due to various reasons, and when the blood flow impacts, the intima ruptures and blood enters the middle layer to form a coarctation, or due to the rupture of the trophoblastic vessels in the arterial wall resulting in an intramural hematoma that gradually expands to the proximal and/or distal end to form an aortic coarctation. Daily precautions for patients with aortic coarctation 1, aortic coarctation patients diet is light, low-fat, high protein, balanced nutrition can be, without excessive supplementation, prevent overheating and cold colds, do not suddenly drink a lot of water or drinks, which will increase the burden on the heart, while increasing the burden on the blood vessels. 2, aortic coarctation patients should not be excited, excitement and sadness are not good, so do not participate in the intense sports not only do not even look, unless you can maintain a quiet state of mind. The above describes the etiology and pathogenesis of aortic coarctation, I hope it will be helpful for those who need it.