The last few days have been rainy and cold, and there has been a significant increase in the number of patients with epiglottic neuritis in the outpatient clinic. If there are any mistakes, please point out in time. The supragluteal neuritis, also known as the supragluteal nerve injury. According to statistics, about 40-60% of acute clinical soft tissue injuries in the lumbar region are due to “displacement” of the superior gluteal nerve in its travels, which is called “tendon out of the groove” in Chinese medicine, and can generally be treated satisfactorily by manipulation with drugs. Anatomical structure: The superior gluteal nerve is a combination of the posterior lateral branches of the spinal nerves of the lumbar 1 to 3; the superior gluteal nerve is a lateral branch of the posterior branches of the lumbar nerves of the 1st to 3rd lumbar nerves, which penetrates the deep fascia parallel to the iliac crest at the intersection of the line between the greater trochanter and the third lumbar vertebrae, and is distributed in the skin of the buttocks, which is not easily palpable. The posterior branches penetrate on the surface of the lumbar dorsal fascia and form the gluteal epicutaneous nerve vascular bundle, which crosses the iliac crest and enters the lobulated fatty connective tissue of the upper gluteal region, and at the ventral edge of the gluteus maximus muscle, the gluteal epicutaneous nerve becomes lobulated with the layered fatty connective tissue, and the gluteal epicutaneous nerve splits into many fine branches to innervate the gluteal fascia and skin tissue of the corresponding area. Pathogenesis: When exercising, especially when the body is rotated from side to side or bent over rapidly, the nerve is easily damaged in the section below the iliac crest and undergoes subtle changes in its anatomical position, deviating from its original position to form a “tendon out slot”. The nerve itself and its surrounding soft tissues become aseptically inflamed. In acute cases, acute congestion and edema may occur; in chronic cases, thickening, enlargement, and blunting of the nerve itself may occur, along with fibrosis and connective tissue hyperplasia of the surrounding soft tissues. When the back muscle fascia is tense when bending and sitting, the local tension increases, which increases the stimulation of the gluteal epicutaneous nerve, especially in acute injury, which can produce severe lumbar and hip pain, and at the same time cause reflex lower limb pain by stimulating the posterior branch of the spinal nerve to the center, and the patient often feels pain in the lumbar and affected lower limbs (the pain generally does not exceed below the knee joint, with the hip, hip and inner thigh being the most important). Clinical manifestations: Most of the patients have a history of “flashing” and “twisting” of the waist and hip, and pain in the waist and hip on one side, which is stabbing, aching or tearing-like pain, and the pain is more intense in the acute stage, and there may be pulling-like pain in the lower extremities, but mostly not in the knee. When changing from a sitting position to an upright position, the lumbar region feels “unable to use force” and cannot stand up or sit down directly, but needs assistance to stand up. (Personally, I think that the pain in patients with epiglottic neuritis is generally on one side of the waist and ipsilateral hip or lower limb pain, rarely accompanied by pain in the posterior middle of the waist; whereas in patients with lumbar disc herniation, in addition to the general symptoms, the pain generally includes pain in the posterior middle of the waist. The identification can be drawn from). In general, patients with gluteal epicutaneous neuritis have a positive straight leg raise test of the lower limb on the healthy side, but no neurogenic signs. Treatment: 1. Manipulative treatment. 2, Topical NSAIDs + oral NSAIDs. 3.Local closure therapy. Note: 1, avoid hot compresses, hot compresses in acute patients can cause rapid aggravation of symptoms. 2, avoid repeated massage, pressure, can aggravate edema, hyperplasia.