Clinically, we often encounter patients with inflammation around one of their teeth, which needs to be extracted as the cause tooth, but due to the infection, they are reluctant to extract it or the doctor does not give it to them, some of them even delay it for months. Not only is it painful and costly for the patient, but sometimes it can also be life-threatening due to serious after-effects. So, what should be done with such inflammatory areas of focal teeth? The best way to deal with such cases is undoubtedly to actively control the inflammation. In the acute inflammatory phase, the decision to extract the focal tooth should be based on the patient’s general condition (e.g., presence of sepsis), the ease of surgery (size of trauma), etc. If the patient’s systemic condition is poor or the surgery is complicated or damaged, extraction may aggravate or spread the inflammation and should be postponed. On the contrary, if the patient’s general condition is good and the surgical damage is small, extraction of the focal tooth under the effective control of sensitive antibacterial drugs at this time will facilitate the drainage of pus, so that the severe pain can be rapidly relieved and the inflammation can be controlled and rapidly improved or cured. The focal tooth itself is the focus of infection, the source of inflammation, and the culture medium of bacteria. Infiltration of inflammatory cells will cause local capillary congestion and increased pain. If we can seize the opportunity of treatment, improve anesthesia and surgical methods, extract the focal tooth that can no longer be saved in time, reduce the local tissue pressure and toxic reaction, and help to limit the inflammation, we can shorten the course of the disease and reduce the occurrence of complications. If the stage of inflammation development is overly considered, and it is considered painful and inappropriate to extract teeth during the acute inflammation period, the inflammation continues to develop, destroying the surrounding tissues, forming fistulas in the oral cavity and on the skin, and even forming diffuse osteomyelitis and sepsis, the disease will be delayed and even life-threatening. Even if a large number of sensitive antibacterial drugs are used, they can only control the acute inflammation and make it chronic because the lesions are not going. As soon as the resistance is lowered, the infection will come on acutely again.