Cheek and upper chest congestion is the main pathological change of renal syndrome hemorrhagic fever, which is clinically characterized by fever, hypotension, hemorrhage, and renal damage. So what are the common tests for cheek and upper chest congestion? Let’s take a look below. (1) Routine examination (1) blood routine The total number of white blood cells is normal or low in the early stage, but after 3 to 4 days, it is obviously increased, mostly in (15-30) × 109/L. Heterotypic lymphocytes can appear in 1 to 2 days of illness, and increase day by day, generally 10% to 20%, partly up to 30% or more; platelets are obviously reduced, the lowest in the period of hypotension and oliguria, and there are heterotypic and meganuclear platelets appear, polyuria in the late stage The platelet count starts to recover. Red blood cells and hemoglobin start to rise in the febrile period, gradually increase in the hypotensive period, rise significantly in patients in the shock period, and fall in the oliguric period, and their dynamic changes can be used as important indicators to determine hematoconcentration and hemodilution. (2) Urine routine Significant urine protein is an important feature of the disease and the earliest manifestation of renal damage. The urine may also contain red blood cells, tubular or membranous material (a mixture of clot, protein and necrotized epithelial cells). 2.Blood biochemical examination (1) Urea nitrogen and creatinine are mildly or moderately increased during hypotensive shock. It peaks during the oliguric phase to polyuric phase, and then gradually decreases, the degree and magnitude of increase is proportional to the condition. (2) Electrolytes Blood potassium may decrease during the febrile period and remain low during the shock period, then increase to high potassium during the oliguric period and decrease again during the polyuric period. However, hypokalemia may also occur during the oliguric phase. Sodium and chloride decrease throughout the course of the disease, most significantly during shock and oliguria. Blood calcium also decreases throughout the course of the disease. (3) Carbon dioxide binding capacity decreases in the late febrile stage, and is obvious in the hypotensive shock stage, and also decreases in the oliguric stage, and gradually returns to normal in the polyuric stage. 3, coagulation function examination Coagulation factors are heavily depleted, platelets are decreased, prothrombin and partial thromboplastin time is prolonged, and fibrinogen is decreased. Secondary hyperfibrinolysis is manifested by prolonged prothrombin coagulation time, increased fibrin degradation material and shortened euglobulin lysis time. The plasma fisetin paraclotting test (3P test) is positive.