Intrauterine adhesions (IUA) are partial or total adhesions and occlusions of the uterine cavity following damage to the basal layer of the endometrium due to various factors. This leads to clinical phenomena such as abnormal menstruation and abnormal fertility in patients. Menstrual abnormalities include: secondary decreased menstrual flow, amenorrhea, cervical canal adhesions leading to irregular menstrual flow, and periodic lower abdominal pain. Usually, any factor that causes endometrial disruption can cause uterine cavity adhesions, and their occurrence is associated with trauma, pregnancy, infection and other factors. According to statistics, about 90% of uterine adhesions are related to pregnancy and are commonly seen after early abortion or curettage. Secondly, placental residues after full-term pregnancy or induction of labor, and uterine adhesions often occur after uterine cavity removal. During pregnancy, due to the softness of the uterine wall, it is not easy to control the depth when scraping the uterus, or excessive scratching of the uterine cavity, excessive negative pressure and time during suction; the endometrial basal layer will be destroyed, resulting in postoperative cervical adhesions; in addition, the surgical instruments repeatedly enter and exit the uterine cavity during surgery, irregular dilatation of the cervix, etc. may aggravate the damage and increase the chance of postoperative cervical adhesions. In addition, non-pregnancy-induced cervical adhesions account for about 10% of cases, such as endometrial tuberculosis, after myomectomy, and after repeated diagnostic scrapings. In recent years, with the increase of painless abortions, the number of patients with uterine adhesions has been increasing. In recent years, with the development of hysteroscopic techniques, transcervical resection of adhesions (TCRA) allows targeted separation or incision of uterine adhesions under direct vision and has become the standard method for the treatment of uterine adhesions. However, the prevention of re-adhesions after TCRA remains a clinical challenge. Mild cavity adhesions can be corrected surgically to restore the shape of the uterine cavity. However, in patients with moderate to severe cavity adhesions, the endometrial basal layer is more severely damaged and the regenerative capacity of the endometrium and glands is low; therefore, even if the cavity shape is restored through cavity adhesion surgery and various methods are given to promote endometrial growth after surgery, the clinical prognosis is not ideal because of the thin endometrium, poor growth, poor cavity tolerance, and the high incidence of re-adhesion. Patients with severe cavity adhesions may not be cured by surgical treatment, and some patients may even suffer from lifelong infertility as a result. In conclusion, the clinical prognosis of patients with cavity adhesions is closely related to the extent and scope of the preoperative cavity lesions and the area and growth of the residual endometrium. Finally, most hysteroscopists have seen too many “abortions at 20 and infertility at 30”. It is strongly recommended that women of childbearing age choose an appropriate form of contraception, avoid unplanned pregnancies, avoid the easy choice of abortion, and avoid the occurrence of repeated multiple abortions.