Herpes zoster is an acute viral skin infection caused by infection with the varicella-zoster virus that shifts from a latent to an outbreak proliferative state, and its onset has multiple triggers. Herpes zoster is caused by an attack of the infected varicella-zoster virus. The virus may be first contracted from outside or may be latent in the body itself. In first-time infections, the virus is usually transmitted to the bloodstream through the mucous membranes of the respiratory tract and transferred through the bloodstream to the dorsal ganglion roots by an outside infection. Since most patients have the first attack with clinical manifestations of chickenpox and mild symptoms, when they are cured, the virus cannot be completely removed from the body and can reappear later. The reason for re-emergence is that the infected virus that has not been completely cleared is latent in the dorsal ganglion roots of the person, and when the person has a trauma or is sick with fatigue, the body’s immunity decreases dramatically at some point and the latent virus becomes active again, multiplying in large numbers and triggering a skin disease, namely herpes zoster. During a herpes zoster attack, the patient may experience a low fever and malaise, and the skin may develop an erythematous papule that rapidly turns into a blister within a short period of time. Because the virus is latent in different nerve sites, the blisters tend to be distributed in bands along the nerves and can be treated with antiviral medications, mostly taken internally. Herpes zoster is caused by an attack of the varicella-zoster virus from an external infection or a relapse after latent in the body, and the symptoms are more pronounced and need to be treated.