Herpes zoster is a disease caused by the varicella-zoster virus, which is also commonly known as the “crosstalk dragon” and the “turnstalk dragon”. The varicella-zoster virus mostly affects children and the elderly, and the virus can be spread among children and the elderly through droplets and contact with herpes fluid, resulting in chickenpox and shingles, respectively. When the autoimmune system is low, such as when traveling, before exams or staying up late, drinking a lot of alcohol, having a cold or fever, having a malignant tumor or chemotherapy, the herpes zoster virus latent in the posterior root ganglion of the cremaster grows and multiplies, causing inflammation and necrosis in the affected ganglion, thus causing neuralgia. At the same time, the virus that invades the peripheral nerve fibers spreads to the corresponding skin causing segmental blisters. The disease is most common in spring and autumn, and usually begins with prodromal symptoms such as mild fever, fatigue, loss of appetite, and other general discomfort, followed a few days later by the appearance of irregular erythema along the nerve distribution, followed by clusters of blisters the size of corn to green beans, accompanied by pain. However, some patients only show erythematous blisters without neuralgia, which is often misdiagnosed as eczema and papular urticaria, while some patients only show neuralgia without rash in the early stage of the disease, which is often misdiagnosed as intercostal neuritis and acute abdominal disease, thus delaying the diagnosis and treatment of the disease and leaving posterior neuralgia. Herpes zoster is usually treated with a combination of analgesia, antiviral, nerve nutrition, immunosuppression and lesion closure, where early administration of small doses of hormones can reduce the occurrence of postherpetic neuralgia.