The etiology of aortic coarctation is unclear, and is mainly related to increased aortic wall tension, structural abnormalities of the aortic wall, and other associated morbidities such as pregnancy. 1. Increased aortic wall tension: including hypertensive disorders, aortic constriction, aneurysms, congenital aortic valve disease, and other factors. 2. Destruction of aortic wall structure: Including hereditary connective tissue diseases such as equine syndrome and Turner syndrome, aortic inflammatory diseases, atherosclerosis and its ulcers. 3. Other related factors: including pregnancy, advanced age, polycystic kidney, blunt or medical injury. Various factors lead to destruction or necrosis of the middle layer of the aorta, increasing the transverse shear force of the vessel wall, resulting in tearing of the intima, and the formation of a false lumen between the walls that communicates with the true lumen of the aorta through the rupture to form an aortic coarctation. If you feel unwell, it is recommended that you seek prompt medical attention.