Herpes zoster is an acute viral infection that affects mainly middle-aged and elderly people. The causative agent is varicella-zoster virus (VZV), which mostly infects humans during childhood and manifests clinically as chickenpox. When the systemic specific immune mechanism is established in varicella patients, most of the VZV is cleared out of the body, and only a very small amount of VZV is stored in the dorsal root ganglion of the crestal medulla or brain ganglion, in a non-replicating latent state, coexisting with the specific immune formation in the host. When the cellular immune status of the body decreases, such as in elderly patients, patients receiving immunosuppressive therapy, and HIV patients, the VZV hosted in the ganglia is activated again and replicates in large numbers and enters the dermatomes along the nerve roots, forming clusters of blisters and pustules with a typical unilateral distribution and causing a series of complications. The highest incidence is postherpetic neuralgia, which can persist for months to years after the lesions have subsided, mostly in middle-aged and elderly people. Due to the unknown cause and limited treatment, it seriously affects the quality of life of middle-aged and elderly people, causing them great pain and imposing a great burden on society. The incidence of herpes zoster in the general population is 10% to 20%, and about 50% of herpes zoster occurs in people over 60 years of age, while about 50% of people over 85 years of age may develop herpes zoster. The annual incidence of herpes zoster gradually increases with age, with 11-29 persons/(year/million) under 50 years of age likely to develop herpes zoster; 46 and 69 persons/(year/million) in the 50-59 and 60-69 years of age groups, respectively; and 95 and 109 persons/(year/million) in the 70-79 and 80-89 years of age groups, respectively. The increase in the incidence of herpes zoster with age may be related to the progressive deterioration of cellular immune function in the elderly. The incidence of herpes zoster is significantly higher in patients with AIDS and in those taking long-term immunosuppressive drugs. People with normal immune function are also susceptible to herpes zoster in cases of exertion, viral infection, and chronic emotional depression. A young man was treated for herpes zoster after staying up late watching football for several days during the European Cup and having an uncontrolled diet. Neuralgia can occur in 60% to 90% of patients during the acute phase of a herpes zoster attack. The pain can be caused by direct stimulation of the peripheral sensory nerves in the lesion area by a large number of inflammatory transmitters produced by the marked inflammatory response in the lesion area, by direct destruction of nerve axons and nerve cells by intense inflammation, or by nerve damage caused by post-inflammatory neuronal cell bleeding. Neuralgia in the acute phase usually worsens with the progression of the lesion and decreases after the lesion is gradually absorbed. In addition to acute neuralgia, it may be accompanied by abnormal nociception and nociceptive hypersensitivity. Although the presentation of acute pain varies widely from patient to patient, most of the pain decreases significantly or disappears several days after the lesions are resolved. The severity and duration of pain in the acute phase correlates significantly with postherpetic neuralgia, a complication of herpes zoster. The goals of herpes zoster treatment are to accelerate herpes resorption, reduce the intensity and duration of pain, and decrease the incidence of complications. Current treatment consists mainly of antiviral therapy and glucocorticoid therapy. Postherpetic neuralgia is the most common complication of herpes zoster, which occurs in middle-aged and elderly people, and has no effective treatment other than symptomatic analgesic treatment because of its long duration and severe pain, which seriously affects patients’ work and quality of life. There is still no uniform definition of postherpetic neuralgia, but the more widely accepted definition is pain lasting for more than 1 month after clinical cure of acute herpes zoster. According to this definition, the incidence of postherpetic neuralgia is 19.2% of the population with shingles and increases with age. The average age of onset of postherpetic neuralgia is 67 years, and the incidence of herpes zoster is approximately 14 times higher in those over 50 years of age than in those under 50 years of age. There is also a relationship between the incidence of postherpetic neuralgia and the site of herpes zoster, with ocular herpes zoster having a higher probability of postherpetic neuralgia than the trunk. In addition, severe pain during the acute phase of herpes zoster, painful prodromal symptoms before the onset of herpes, and fever over 38°C during the acute phase are all risk factors for the development of postherpetic neuralgia. Acupuncture has a long history of treating herpes zoster, and both the herpes phase and post-neuralgia phase can be treated with acupuncture. Acupuncture methods can effectively reduce the intensity of pain, shorten the duration of pain and reduce the incidence of postherpetic neuralgia. At present, there is still no satisfactory treatment for postherpetic neuralgia, many patients need to rely on drugs (and some drugs such as Premarin, expensive) to relieve pain, in addition to long-term medication and side effects, and even a small number of patients can not find effective pain relief, long-term pain suffering suffering, the quality of life is low, over time patients are depressed, depression, loss of confidence in life In the long run, the patients are depressed, depressed, lose confidence in life, and even have suicidal tendencies. We have achieved good results in the clinical treatment of postherpetic neuralgia with acupuncture. The methods commonly used in clinical practice are fire acupuncture, milli-needle peri-prick, red light, etc. Some of them are coupled with acupuncture point injection of nerve-nourishing drugs and acupuncture cupping. Many people who have suffered from post-herpetic neuralgia for months or even years have been cured, usually with 10 to 20 treatments. If you have shingles or post-neuralgia, you may want to try acupuncture. A few common questions about shingles: 1. Is shingles contagious? Herpes zoster patients with blistering lesions contain a large number of viruses that can spread outward to the skin surface and air as the herpes breaks, and children or adults without immunity can be infected after contact through the whistle and can develop chickenpox. The majority of these are caused by the reactivation of their own viruses, but we do see some patients with a clear history of contact before the onset of the disease, so we do not rule out the possibility of contact transmission of herpes zoster. 2. Can herpes zoster recur after being cured? The majority of patients who are cured of shingles gain lifelong immunity to the virus and rarely relapse. However, a few people with poor immunity or chronic illnesses that cause a decline in immunity can get shingles a second time, or even multiple times. 3, herpes zoster care points (1) diet should be light, easy to digest, eat more fresh fruits and vegetables, drink more water, eat less fried, fried products, avoid fish, shrimp, crab, dog meat, mutton and other hairy products, avoid spicy stimulating food, no smoking and alcohol. (2) During the treatment period, you should take a good rest, avoid straining and getting angry.