G6PD deficiency is caused by a mutation in the G6PD gene, resulting in a decrease in the activity of the enzyme and the destruction of red blood cells that cannot resist oxidative damage, causing hemolytic anemia. The clinical manifestations of G6PD deficiency are approximately the same as those of general hemolytic anemia. It is divided into clinical types such as neonatal jaundice, serum sickness, pharmacologic hemolysis, infectious hemolysis, and non-spherocytic hemolytic anemia. The clinical manifestations of the disease vary in severity. Most patients, especially female heterozygotes, usually do not develop the disease and have no conscious symptoms, and some patients may show symptoms of chronic hemolytic anemia. Acute hemolytic reactions such as hemoglobinuria, jaundice and anemia are often induced by consumption of fava beans, taking or exposure to certain drugs and infections. Severe acute hemolytic anemia induced by G6PD deficiency can cause liver, kidney, or heart failure or even death if left untreated due to excessive red blood cell destruction. When G6PD deficiency is not induced, it is the same as normal people and requires no special treatment. The key to prevention and treatment is prevention and strict adherence to the following health prescriptions to prevent attacks. The next step is the administration of small doses of phenobarbital to pregnant women or newborns in late pregnancy, which can effectively reduce the occurrence of neonatal nuclear jaundice. Blood transfusion is the most effective therapy in acute attacks of the disease, followed by correction of acidosis and management of renal failure. Patients with mild to moderate hemolysis are usually treated with rehydration. Health prescriptions for patients with G6PD deficiency: 1. Forbid eating fava beans or raw processed fava beans, and avoid going to fava bean fields during the flowering, fruiting or harvesting season. 2.Prohibit the use of naphthalene-containing stink pills into the closet to repel insects. 3.Prohibited drugs acetanilide, methylene blue, nitroimidazole, furadantin, furazolidone, furacilin, phenylhydrazine, primaquine, paromomycin, pentamidine quinine, sulfanilamide, acetylsulfonamide, sulfapyridine, thiazolone, toluidine blue, SMZ, TNT, etc. 4.Caution drugs paracetamol, felastidine, aspirin, aminopyrine, antipyrine, Antan, vitamin C, vitamin K, chloramphenicol, streptomycin, isoniazid, sulfadiazine, sulfaguanidine, sulfisoxazole, chloroquine, colchicine, diphenhydramine, levodopa, phenytoin sodium, procainamide, ethacrynic acid, quinidine, quinine, SM, TMP, eugenol, etc. 5, Infectious causative viral hepatitis, influenza, pneumonia, typhoid, mumps, etc. 6.Any symptoms such as fever, abdominal pain, vomiting, yellow or pale face, yellow-brown or dark red urine after infection or contact/consumption of the above foods or drugs for several hours or days are acute hemolytic reactions and should be seen immediately in the emergency department of a hospital! G6PD deficiency does not require special treatment when it does not develop without a cause, just like normal people. It is also not easy to detect. The key to prevention and treatment is prevention and strict adherence to health prescriptions to prevent attacks. Blood transfusion is the most effective therapy in acute attacks of this disease.