In carbon monoxide poisoning, the first organ to be damaged is the brain, as carbon monoxide easily combines with hemoglobin in the blood to form carboxyhemoglobin. The most serious effect of carboxyhemoglobin on the cerebral cortex is the clinical manifestation of cerebral hypoxia, and the severity of hypoxia is proportional to the saturation of carboxyhemoglobin. In mild carbon monoxide poisoning, there will be headache, weakness, vertigo, difficulty in breathing during labor, and even transient syncope during labor, and generally clear consciousness. If moderate to severe poisoning occurs, the patient will show cherry red lips, nausea, vomiting, blurred consciousness, and even coma. When the concentration of carboxyhemoglobin in the body reaches more than 50%, the patient will have cerebral edema, pulmonary edema, myocardial damage, cardiac arrhythmia, respiratory depression, and cause death.