Auditory nerve sheath tumor originates from the auditory nerve sheath and is a typical nerve sheath tumor, accounting for 7% to 10% of intracranial tumors, 93% of intracranial nerve sheath tumors, and 72% of pontocerebellar horn tumors, with a prevalence of 30-60 years old and a slightly higher incidence in women than men. The symptoms can be mild or severe, which are mainly related to the starting site, growth rate, development direction, tumor size, blood supply and whether there is cystic change. The duration of symptoms may vary from a few months to a few years, usually lasting from 3 to 5 years. Most patients present with symptoms of auditory nerve sheath tumor, including dizziness, tinnitus and hearing loss, which may occur simultaneously or sequentially. Tinnitus is high-pitched, resembling the sound of a cicada or a siren, and is continuous, often accompanied by hearing loss. Deafness is more important, as deafness is reported to exist in 85.2% to 100% of cases, while tinnitus exists in only 63% to 66.9% of cases, according to relevant data. Dizziness is often unnoticed by patients and physicians because it is mild and is not accompanied by nausea and vomiting. Deafness, on the other hand, is an objective sign and can be detected. If unilateral deafness is not accompanied by tinnitus, it is often not noticed by the patient, and hearing loss is occasionally noticed when listening to the telephone, or until complete deafness or other related neurological symptoms occur. In addition to the above-mentioned neurological impairments, due to the influence of national health concepts and medical habits, most patients often do not remember to come to the hospital until they have symptoms of increased intracranial pressure. Increased intracranial pressure is one of the common clinical features of auditory nerve sheath tumors. The early and late appearance of the symptoms of increased intracranial pressure is related to the size, growth rate and location of the tumor. Generally speaking, the larger the size of tumor, the more obvious the symptoms of increased intracranial pressure. However, for medial tumor, because the tumor is close to the midline, although the tumor size is not large, the cerebrospinal fluid circulation is affected in the early stage, producing obstructive hydrocephalus, and the symptoms of increased intracranial pressure can appear in the early stage of the disease and are more significant. There are also some symptoms and signs that are easy to appear when the tumor grows to a larger size: 1 Cerebellar dysfunction: the cerebellar hemispheres are deformed by tumor compression, and sometimes some tumors protrude into the cerebellar hemispheres. The cerebellar dysfunction is different from that of cerebral hemispheres, and the ataxic movement disorder caused by the damage varies greatly from almost unnoticeable in mild cases to bedridden in severe cases, which may be related to the degree of lesion development.2 Brainstem signs: Brainstem signs are caused by the tumor growing medially and compressing the corresponding structures of brainstem. In patients with medial type of auditory nerve sheath tumor, because the tumor growth point is close to the brainstem, the brainstem symptoms appear earlier and are more severe, while in most patients, the brainstem symptoms appear relatively late, and they are mostly seen in patients with large and giant type of auditory nerve sheath tumor. Some patients may develop bilateral pyramidal fasciculus signs.3 Brain nerve damage: In addition to the upward reaching of the trigeminal nerve mentioned above, the downward development of the tumor may compress the Ⅸ, X, and D. cerebral nerves, as the nerves in this group are displaced posteriorly and inferiorly, resulting in the late appearance of clinical symptoms and signs, and the degree of damage is also mild. Choking and difficulty in swallowing when eating. If there is collateral nerve damage, it can be manifested as weakness of neck rotation and shoulder shrugging on the affected side, and atrophy of sternocleidomastoid and trapezius muscles on examination. The posterior cerebral nerve damage can be unilateral or bilateral, or the lesion can be focused on the opposite side with slightly less severe symptoms. The hypoglossal nerve is located medially and is not easily damaged by compression. The damage can be seen as atrophy of the tongue muscle on the affected side, with fibrillation of the tongue muscle and deflection to the diseased side when extending the tongue. Therefore, we should be alert if we encounter unilateral tinnitus, inaudible phone calls, unstable walking or typical signs of increased intracranial pressure in our lives. It is important to visit a specialist in a timely manner and have an MRI and other special tests done if necessary.