It is caused by early closure of the sagittal suture alone, and is the most common cephalic malformation in premature closure of the cranial suture, accounting for about 40% to 70% of cases. Premature closure of the sagittal suture restricts the development of the head laterally, i.e., it expands anteriorly and posteriorly, resulting in an anterior-posterior elongation of the cranial vault and left-right stenosis, which results in a saddle-like deformity of the skull, with ocipital and frontal pole overstretching. The frontal bone can be very high, resulting in a pear-shaped forehead due to narrowing of the intertemporal fossa. Early closure of the sagittal suture causes navicular head deformity, males account for the majority of men, the ratio of men to women is 4:1. occasionally there is a family history. 1, navicular head deformity: also known as long head deformity, caused by the early closure of the sagittal suture alone, is the most common cranial deformity in the early closure of the cranial suture, accounting for about 40% to 70%. Premature closure of the sagittal suture restricts the development of the head to the side, i.e., it expands forward and backward, resulting in an anterior-posterior elongation of the cranial vault and narrowing of the left and right sides, which results in a saddle-like deformity of the skull, with an over-expansion of the occipital and frontal poles. The frontal bone can be very high, resulting in a pear-shaped forehead due to narrowing of the intertemporal fossa. Early closure of the sagittal suture results in navicular head deformity, and males account for the majority of cases, with a male-to-female ratio of 4:1. There is occasionally a family history of this condition. 2. Crouzon craniosynostosis: Crouzon craniofacial dysplasia or Crouzon craniofacial stenosis is also known as Crouzon craniofacial dysplasia. It was first reported by Crouzon in 1912. The main features of this disease are: (1) Huge cranial cap and early closure of cranial sutures, with the coronal suture and herringbone suture closure being the most common, and the cranial head bulging due to the ossification of the fontanel protruding from the anterior fontanel. (2) Relative protrusion of the normal mandible compared to the tiny maxilla, facial nasomandibular retraction, resulting in occlusal inversion and to some extent pseudo-convex jaw deformity. (3) The nose is excessively protruding as a hawk’s beak nose, the orbital wall is pushed forward, the supraorbital rim is retracted due to the short head deformity, and the infraorbital rim is also retracted due to the jaw retraction, which results in the formation of extreme eye protrusion, and this protrusion, together with the orbital widening, creates the frog’s eye of Crouzon’s disease. The patient may be accompanied by oculomotor paralysis. (4) Most have a hereditary and family history of the disease, also known as hereditary cephalofacial dysostosis. (5) The disease may be associated with increased intracranial pressure, loss of vision and mental retardation. The diagnosis is not difficult for those who present with typical manifestations of cephalic deformity. However, when cranial deformity is found after birth, it is often misdiagnosed as a result of childbirth. If the cranial deformity does not disappear for a certain period of time after birth, a cranial X-ray radiography should be performed. The main manifestations are increased density at the cranial bony sutures, calcium deposits, and sometimes signs of increased intracranial pressure such as increased pressure traces in the cerebral gyrus and decalcification of the posterior bed protrusion. This disease must be distinguished from microcephaly, microcephaly is due to the primary brain development disorder, the skull did not increase due to microcephaly, is not the early closure of the cranial sutures to limit the development of brain tissue, the cranial sutures are also closed, for the secondary craniosynostosis. Patients often do not have increased intracranial pressure, mental retardation is more obvious, X-ray examination of the density of the bone suture may be normal or there is no cerebral gyrus pressure trace increase and other signs of intracranial hypertension.