Most patients with medulla oblongata infarction will recover in about half a month if they are given active treatment, and some patients may have sequelae that can be followed by rehabilitation. The medulla oblongata is a critical infarct because the medulla oblongata is the respiratory and cardiac center. If the medulla oblongata is large and edematous, the patient can suddenly suffer respiratory and cardiac arrest. If the medulla oblongata has a linguopharyngeal nerve, the patient may have dysarthria, choking and coughing, etc. If necessary, the patient should be given an indwelling gastric tube for feeding. Patients with medullary infarction may also develop Horner’s syndrome, which is characterized by smaller eye fissures on one side of the face, absence of sweating on the face, smaller pupils, and numbness of the opposite limb. In the acute stage of medullary infarction, patients should be given active antiplatelet aggregation, anticoagulation or fibrin-lowering treatment, and at the same time, the patient should be given injectable thromboxane and shuxin injection to activate blood stasis and promote blood circulation. If necessary, give the patient with mannitol, glycerol fructose and other drugs for dehydration and reducing intracranial pressure to avoid life-threatening edema in severe cases.