Shoulder coagulation occurs due to the complex anatomical structure of the shoulder joint. The bony structure of the shoulder joint is mainly composed of the glenoid of the scapula and the humeral head, which is a typical ball and socket joint, and is also the joint with the greatest mobility and flexibility in the human body. The shoulder joint in a broad sense includes the glenohumeral joint, acromioclavicular joint, sternoclavicular joint and scapulothoracic wall joint, which has an extremely complex structure. The ligaments surrounding the shoulder joint are mainly composed of the rotator cuff, the tendon of the long head of the biceps muscle, the rostro-humeral ligament, the glenohumeral ligament and the transverse humeral ligament. The rotator cuff is particularly important in the development of shoulder condensation and consists of the tendons of the supraspinatus, infraspinatus, teres minor, and subscapularis muscles that originate from the scapula and end at the upper end of the humerus. The broad concept of shoulder dystocia includes rotator cuff injuries, tendonitis of the long head of the biceps, glenohumeral arthritis, subacromial bursitis, supraspinatus (infraspinatus) tendonitis and its tenosynovitis, rostro-humeral ligament inflammation, and acromioclavicular arthritis, among many other disorders. Narrowly defined, it is only referred to as “frozen shoulder” or “fifty shoulder”. Due to the wide range of lesions and complex structures involved in shoulder coagulopathy, covering almost all parts of the shoulder joint in the broad sense, coupled with the rich ligaments of the shoulder joint and the intricacies of the nerves and blood vessels, its pathogenesis is still in the exploratory stage, and there is no uniform conclusion so far.