The survival time of cerebellar atrophy is closely related to the specific cause, age, the speed of progression, and other concomitant diseases, and it varies in different cases. Cerebellar atrophy is often characterized by ataxia, balance dysfunction, and dysarthria. Cerebellar earthworm lesions are characterized by unstable standing, wobbly gait, and swaying from side to side like drunkenness, hence the name drunken gait, but limb ataxia is not obvious, and nystagmus is mild or not obvious. Cerebellar hemisphere lesions may present with ipsilateral limb ataxia, horizontal nystagmus and cerebellar speech, such as slurred or explosive speech. Cerebellar atrophy can be seen after cerebral infarction or hemorrhage, genetic, degenerative diseases, and also in poisoning. Cerebellar atrophy generally progresses slowly and there is a lack of effective treatment, most of which can last for several years, and there is no exact clinical value for the exact duration. Once cerebellar atrophy is detected, the cause should first be identified and attention should be paid to functional exercise to slow down the progression of the disease.