In recent years, as the pace of life continues to increase, as well as the frequency of computer and video game consoles, the incidence of cervical disc herniation continues to rise, the age of onset is also gradually younger, cervical vertigo in the whole spine disease patients accounted for a growing proportion, has seriously affected people’s life and work. The clinical manifestations of cervical disc herniation are often characterized by chronic spinal cord or nerve root compression damage and corresponding symptoms. In practice, it is observed that the cervical nerves are not as long as the lumbar nerve roots in the spinal canal and the degree of freedom is large, the protruding cervical intervertebral discs not only compress the spinal cord, but also often compress the neighboring nerve roots, which is the reason for the diversity and complexity of the clinical manifestations of cervical disc herniation. This is the reason for the diversity and complexity of the clinical manifestations of cervical disc herniation. Generally speaking, there are spinal cord type, nerve root type, vertebral artery type and mixed type in the clinical classification. Cervicogenic vertigo is the vertebral artery or cervical sympathetic nerve compression and stimulation by the cervical lesion, causing the vertebral-basilar artery blood supply is insufficient, the brainstem, cerebellum and vestibular system ischemia and hypoxia and caused by vertigo as the main clinical syndromes, in which the sympathetic nerve stimulation caused by arterial spasms and the occurrence of the disease of the argument is increasingly being paid attention to. The etiology of discogenic vertigo In 1926, Bam-Lieou pointed out that cervical degeneration could stimulate the sympathetic nerves to cause spasm of the vertebral arteries leading to vertigo In 1933, Dek1ey et al. referred to the vertebral basilar artery insufficiency of blood supply (vertebm-basilar is-chemia) as a series of clinical syndromes caused by vertigo, headache, nausea and vomiting, weakness of the limbs, and sudden collapse. Cervical vertigo. Subsequently, with the in-depth study of vertebral artery blood supply insufficiency caused by cervical spine degeneration, the diagnostic terminology has been updated, such as Ba-eou syndrome, vertebral artery ischemia syndrome, vertebral artery-type cervical spondylopathy, and so on. Some people put forward mechanical compression, vertebral artery spasm, sympathetic nerve provocation, congenital developmental malformation of the vertebral artery, acquired loss of compensation, etc., but a single theory is difficult to fully explain the clinical pathogenesis. Regardless of how the expression changes, the common points of its connotation are: ① The disease occurs on the basis of cervical degeneration, and after biochemical and physical examination, multidisciplinary consultation, exclude hypertension, atherosclerosis, cervical spinal cord vascular malformation, atlantoaxial hook and loop deformity, cerebral, auricular, ophthalmologic, and other diseases caused by vertigo. ② Episodes of vertigo are associated with changes in the position of the neck. ③ It is usually accompanied by sympathetic excitatory phase or inhibitory phase symptoms, such as headache, vision, and hearing abnormalities. Vertigo and other accompanying symptoms are intermittent, sometimes severe, sometimes mild, and easy to recur. The causes of cervical vertigo can be summarized as cervical spondylosis, cervical instability, cervical disc herniation, and cervical fracture and dislocation. Among them, cervical disc herniation is the more common cause of cervical vertigo, and patients are often accompanied by cervical nerve root and spinal cord compression symptoms. The herniated discs irritate the dura mater, nerve roots and sympathetic nerves around the arteries and veins during neck movement, and may also directly compress the cervical cord segment of the vestibular spinal cord bundle, causing reflex vertigo. Mild instability of the cervical spine may cause edema of the paravertebral ligaments and muscles due to strain, or squeeze the vertebral artery or provoke the paravertebral sympathetic plexus, which may induce spasm of the vertebral arteries and incomplete blood supply.