Can non-verbal or speech disorders also be epilepsy? A parent responded: My child used to be able to speak well, but recently I found that my child is often dull and seems to gradually dislike talking. Can non-verbal or speech disorders also be epilepsy? Can non-verbal or speech disorders also be epilepsy? Some children who do not speak or have speech disorders are often asked by neurologists to have a 24-hour ambulatory EEG when they visit the hospital. Some parents are very confused and even think that the doctor is not being reasonable in prescribing various tests. In fact, sometimes non-verbal or speech disorders are a clinical manifestation of seizures. There is a classification of epilepsy called acquired aphasic epilepsy (LKS), which is a rare syndrome characterized by acquired aphasia and seizures. The epilepsy is age-related and is associated with acquired language impairment. Children with LKS often start with a loss of hearing, comprehension, and spoken language, which can be mistaken for “deafness,” and hearing, comprehension, and spoken language impairments are often thought by family members to be developmental delays or premature births at the onset, leading to delays in treatment. Can non-speech or speech disorders also be epilepsy? Acquired aphasia is a necessary symptom of LKS. Children with LKS have normal language development before the onset of the disease, and most have a subacute onset that can be exacerbated by seizures. Aphasia is characterized by auditory aphasia, i.e., loss of the ability to understand the spoken words of others and the inability to carry out simple instructions given by the spoken word, accompanied by varying degrees of decline in numeracy, memory, and written language skills, but retention of the understanding of nonverbal sounds. Aphasia can occur suddenly or gradually, and it can occur before, at the same time, or after the onset of epileptic symptoms. Clinical symptoms fluctuate, and aphasia may improve or worsen spontaneously. Can non-verbal or speech disorders be epilepsy? The pathogenesis of acquired epileptic aphasia is unknown, but many non-specific abnormalities in the pathology have been shown to be possibly associated with acquired epileptic aphasia, including subcortical astrocytosis, encephalitis, myelin loss, chronic viral infections, cerebral arteritis, cysticercosis, arteriovenous malformations, neuronal developmental defects, toxoplasmosis, and temporal lobe tumors.The clinical presentation of LKS is unique and can be easily misdiagnosed. It should be differentiated from speech delay due to cerebral palsy, deafness, autism, benign epilepsy in children with central temporal spikes, and epilepsy with slow-wave sleep-period sustained spikes and slow waves.