Scissor gait of the lower limbs is a clinical manifestation of spastic bilateral lower limb paralysis in cerebral palsy. The scissor gait is due to increased muscle tone in both lower extremities, especially in the extensor muscles, and the lower extremities are over-extended when moving, and the legs are crossed in a scissor shape. A scissor gait in the lower limbs is a sign of cerebral palsy or paraplegia. The common causes of cerebral palsy are as follows: 1. Prenatal factors 1. Embryonic brain development abnormalities such as microcephaly, congenital hydrocephalus, macrocephaly or anencephaly. 2. 2. Trauma to the mother during pregnancy, pregnancy toxemia, diabetes and radiation exposure can affect fetal brain development and cause permanent brain damage. 3.Rubella and toxoplasmosis in early gestation can affect the development of the fetal central nervous system and cause disease. 4. Premature babies and small children, the younger the gestational age, the more incidences. It is related to the incomplete development of the nervous system of preterm infants, easy bleeding and hypoxia. 5. The placenta of premature babies is degenerative and necrotic, causing hypoxemia, resulting in fetal hypoxia. Second, the causes of labor and delivery 1, cerebral hypoxia delivery time is too long, prenatal use of anesthetics, sedatives can inhibit fetal respiration to cause fetal hypoxia, in addition to the umbilical cord around the neck, early abruption of the placenta, anterior can cause fetal cerebral hypoxia. 2, cerebral hemorrhage birth injury, emergency delivery, obstructed labor and hemorrhagic diseases can cause intracranial hemorrhage. 3. Postnatal causes such as neonatal hyperbilirubin-induced nuclear jaundice, meningitis, encephalitis or systemic severe infection-induced toxic encephalopathy, head trauma, carbon monoxide poisoning, etc. can also be diagnosed as sequelae of a disease. The maintenance of normal muscle tone regulation and postural reflexes depends on the dynamic balance between the inhibitory effect of cortical downstream fibers and the excitatory effect of peripheral afferent fibers. Impaired perceptual abilities, such as visual and auditory abilities, can cause mental retardation, damage to the basal ganglia can lead to tardive dyskinesia, and damage to the cerebellum can lead to ataxia. There are two types of specific pathological changes in cerebral palsy: (1) hemorrhagic damage, which can be seen as subventricular hemorrhage or intraventricular hemorrhage, mostly in immature infants less than 32 weeks of gestation, probably due to the relatively high cerebral blood flow, fragile blood vessels, imperfect vascular nerve development and poor ability to regulate cerebral blood flow during this period; (2) ischemic damage, such as cerebral white matter softening, cortical atrophy or atrophic lobar sclerosis, mostly seen in hypoxic (ii) ischemic damage, such as cerebral white matter softening, cortical atrophy or atrophic lobar sclerosis, is most common in infants with hypoxia.