The normal range of liver glutamic transaminase is 8-40 ∪/L, so the patient’s glutamic transaminase 44U/L is not a normal phenomenon, which is a high performance. Its elevation is generally considered to be related to physiological or pathological factors. 1. Physiological factors: prolonged late night, excessive alcohol consumption, and taking drugs with liver damage can increase the burden on the liver, resulting in a mild increase in AST. During the period of elevated AAT, patients will not have obvious clinical symptoms, and the elevated AAT can be improved by self-conditioning, so it is recommended to pay attention to observation. 2. Pathologic factors: (1) Liver diseases: during the onset of liver diseases such as hepatitis, cirrhosis, liver cancer, etc., the stimulation of disease-causing factors can lead to a large number of liver cell necrosis, resulting in the release of liver cells into the blood to trigger the phenomenon of high alachlor aminotransferase; (2) Heart diseases: during the onset of heart diseases such as myocarditis and myocardial infarction, patients may suffer from myocardial ischemia and necrosis, resulting in the release of glutamine transaminase from cardiac myocytes into the bloodstream, leading to the phenomenon of high glutamine transaminase. In addition, dermatomyositis, myasthenia gravis, and bone cancer and other skeletal muscle diseases can also cause high AST. When AST is elevated, it should be analyzed in conjunction with AST and overall liver function. It is recommended that patients actively go to regular hospitals, under the guidance of the doctor for targeted treatment.